Inhibiting the Migration of M1 Microglia at Hyperacute Period Could Improve Outcome of tMCAO Rats
Published 2016 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Inhibiting the Migration of M1 Microglia at Hyperacute Period Could Improve Outcome of tMCAO Rats
Authors
Keywords
-
Journal
CNS Neuroscience & Therapeutics
Volume 23, Issue 3, Pages 222-232
Publisher
Wiley
Online
2016-12-19
DOI
10.1111/cns.12665
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Treatment with AMD3100 attenuates the microglial response and improves outcome after experimental stroke
- (2015) Helene L Walter et al. Journal of Neuroinflammation
- CXCR4 antagonist AMD3100 reverses the neurogenesis and behavioral recovery promoted by forced limb-use in stroke rats
- (2015) Shanshan Zhao et al. RESTORATIVE NEUROLOGY AND NEUROSCIENCE
- The Ischemic Environment Drives Microglia and Macrophage Function
- (2015) Stefano Fumagalli et al. Frontiers in Neurology
- Pericyte contractility controls endothelial cell cycle progression and sprouting: insights into angiogenic switch mechanics
- (2014) Jennifer T. Durham et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Endogenous Endothelial Progenitor Cells Participate in Neovascularization via CXCR4/SDF-1 axis and Improve Outcome After Stroke
- (2014) Ling Mao et al. CNS Neuroscience & Therapeutics
- Neuroinflammation and M2 microglia: the good, the bad, and the inflamed
- (2014) Jonathan D Cherry et al. Journal of Neuroinflammation
- Postacute Stromal Cell–Derived Factor-1α Expression Promotes Neurovascular Recovery in Ischemic Mice
- (2014) Yaning Li et al. STROKE
- Microglial Responses after Ischemic Stroke and Intracerebral Hemorrhage
- (2013) Roslyn A. Taylor et al. Clinical & Developmental Immunology
- Stromal derived growth factor-1 (CXCL12) modulates synaptic transmission to immature neurons during post-ischemic cerebral repair
- (2013) Agnieszka A Ardelt et al. EXPERIMENTAL NEUROLOGY
- Inhibition of CXCL12 Signaling Attenuates the Postischemic Immune Response and Improves Functional Recovery after Stroke
- (2013) Karsten Ruscher et al. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM
- Sonic Hedgehog (Shh) Regulates the Expression of Angiogenic Growth Factors in Oxygen–Glucose-Deprived Astrocytes by Mediating the Nuclear Receptor NR2F2
- (2013) Yanan Li et al. MOLECULAR NEUROBIOLOGY
- CXCR4 and CXCR7 form a functional receptor unit for SDF-1/CXCL12 in primary rodent microglia
- (2013) J. Lipfert et al. NEUROPATHOLOGY AND APPLIED NEUROBIOLOGY
- Minocycline selectively inhibits M1 polarization of microglia
- (2013) K Kobayashi et al. Cell Death & Disease
- N9 microglial cells polarized by LPS and IL4 show differential responses to secondary environmental stimuli
- (2012) Hong-Cui Liu et al. CELLULAR IMMUNOLOGY
- The Alternative Complement Pathway Propagates Inflammation and Injury in Murine Ischemic Stroke
- (2012) Andrew Elvington et al. JOURNAL OF IMMUNOLOGY
- Microglia/Macrophage Polarization Dynamics Reveal Novel Mechanism of Injury Expansion After Focal Cerebral Ischemia
- (2012) Xiaoming Hu et al. STROKE
- CXCR4 Antagonist AMD3100 Protects Blood–Brain Barrier Integrity and Reduces Inflammatory Response After Focal Ischemia in Mice
- (2012) Jun Huang et al. STROKE
- Temporal pattern of expression and colocalization of microglia/macrophage phenotype markers following brain ischemic injury in mice
- (2011) Carlo Perego et al. Journal of Neuroinflammation
- The Science of Stroke: Mechanisms in Search of Treatments
- (2010) Michael A. Moskowitz et al. NEURON
- Temporal and Spatial Dynamics of Cerebral Immune Cell Accumulation in Stroke
- (2009) Mathias Gelderblom et al. STROKE
- Hypoxia enhances CXCR4 expression favoring microglia migration via HIF-1α activation
- (2008) Xubu Wang et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started