Tumor necrosis factor alpha (TNF-α) blockade improves natural killer cell (NK) activation, hypertension, and mitochondrial oxidative stress in a preclinical rat model of preeclampsia
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Title
Tumor necrosis factor alpha (TNF-α) blockade improves natural killer cell (NK) activation, hypertension, and mitochondrial oxidative stress in a preclinical rat model of preeclampsia
Authors
Keywords
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Journal
HYPERTENSION IN PREGNANCY
Volume -, Issue -, Pages 1-6
Publisher
Informa UK Limited
Online
2020-07-10
DOI
10.1080/10641955.2020.1793999
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Note: Only part of the references are listed.- Natural Killer cells contribute to mitochondrial dysfunction in response to placental ischemia in Reduced Uterine Perfusion Pressure (RUPP) rats.
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- Role of Mitochondrial Dysfunction and Reactive Oxygen Species in Mediating Hypertension in the Reduced Uterine Perfusion Pressure Rat Model of Preeclampsia
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- Restoration of peripheral blood natural killer and B cell levels in patients affected by rheumatoid and psoriatic arthritis during etanercept treatment
- (2014) P. Conigliaro et al. CLINICAL AND EXPERIMENTAL IMMUNOLOGY
- Safety of Anti-TNF Agents During Pregnancy and Breastfeeding in Women With Inflammatory Bowel Disease
- (2013) Javier P Gisbert et al. AMERICAN JOURNAL OF GASTROENTEROLOGY
- Safety of TNF-α inhibitors during IBD pregnancy: a systematic review
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- Control of soluble fms-like tyrosine-1 (sFlt-1) production response to placental ischemia/hypoxia: role of tumor necrosis factor-α
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- Hypertension in Response to Chronic Reductions in Uterine Perfusion in Pregnant Rats
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