Large Extracellular Vesicle-Associated Rap1 Accumulates in Atherosclerotic Plaques, Correlates with Vascular Risks and Is Involved in Atherosclerosis

Extracellular Vesicles: Mechanisms in Human Health and Disease

(2018) Marine Malloci et al.   ANTIOXIDANTS & REDOX SIGNALING

Rap1 promotes proliferation and migration of vascular smooth muscle cell via the ERK pathway

(2018) Qin Li et al.   PATHOLOGY RESEARCH AND PRACTICE

Common carotid artery end-diastolic velocity and acceleration time can predict degree of internal carotid artery stenosis

(2017) David S. Strosberg et al.   JOURNAL OF VASCULAR SURGERY

The TyG index may predict the development of cardiovascular events

(2016) Laura Sánchez-Íñigo et al.   EUROPEAN JOURNAL OF CLINICAL INVESTIGATION

C3G knock-down enhances migration and invasion by increasing Rap1-mediated p38α activation, while it impairs tumor growth through p38α-independent mechanisms

(2016) Neibla Priego et al.   Oncotarget

ERK5 Protein Promotes, whereas MEK1 Protein Differentially Regulates, the Toll-like Receptor 2 Protein-dependent Activation of Human Endothelial Cells and Monocytes

(2012) Kevin Wilhelmsen et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Microparticles from Patients with Metabolic Syndrome Induce Vascular Hypo-Reactivity via Fas/Fas-Ligand Pathway in Mice

(2011) Abdelali Agouni et al.   PLoS One

Harmonizing the Metabolic Syndrome

(2009) K.G.M.M. Alberti et al.   CIRCULATION

Role of the cAMP-binding protein Epac in cardiovascular physiology and pathophysiology

(2009) Mélanie Métrich et al.   PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY

Impact of incomplete DNase I treatment on human macrophage proteome analysis

(2009) Adelina Elena Acosta-Martin et al.   Proteomics Clinical Applications

Endothelial Dysfunction Caused by Circulating Microparticles from Patients with Metabolic Syndrome

(2008) Abdelali Agouni et al.   AMERICAN JOURNAL OF PATHOLOGY

Epac1 is upregulated during neointima formation and promotes vascular smooth muscle cell migration

(2008) Utako Yokoyama et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY