β3-Adrenoceptor redistribution impairs NO/cGMP/PDE2 signalling in failing cardiomyocytes
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Title
β3-Adrenoceptor redistribution impairs NO/cGMP/PDE2 signalling in failing cardiomyocytes
Authors
Keywords
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Journal
eLife
Volume 9, Issue -, Pages -
Publisher
eLife Sciences Publications, Ltd
Online
2020-03-31
DOI
10.7554/elife.52221
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Related references
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- Phosphodiesterase 9A controls nitric-oxide-independent cGMP and hypertrophic heart disease
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- Transgenic Mice for Real-Time Visualization of cGMP in Intact Adult Cardiomyocytes
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- Enhanced Expression of β3-Adrenoceptors in Cardiac Myocytes Attenuates Neurohormone-Induced Hypertrophic Remodeling Through Nitric Oxide Synthase
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- Phosphodiesterase-2 Is Up-Regulated in Human Failing Hearts and Blunts β-Adrenergic Responses in Cardiomyocytes
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- sGCα1 mediates the negative inotropic effects of NO in cardiac myocytes independent of changes in calcium handling
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- Pressure-Overload–Induced Subcellular Relocalization/Oxidation of Soluble Guanylyl Cyclase in the Heart Modulates Enzyme Stimulation
- (2011) Emily J. Tsai et al. CIRCULATION RESEARCH
- Nitric oxide synthase and cyclic GMP signaling in cardiac myocytes: From contractility to remodeling
- (2011) Joanna Hammond et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Caveolae compartmentalise β2-adrenoceptor signals by curtailing cAMP production and maintaining phosphatase activity in the sarcoplasmic reticulum of the adult ventricular myocyte
- (2011) David A. MacDougall et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- 2-Adrenergic Receptor Redistribution in Heart Failure Changes cAMP Compartmentation
- (2010) V. O. Nikolaev et al. SCIENCE
- Loss of T-tubules and other changes to surface topography in ventricular myocytes from failing human and rat heart
- (2009) A. R. Lyon et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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