Non-catalytic ubiquitin binding by A20 prevents psoriatic arthritis–like disease and inflammation
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Title
Non-catalytic ubiquitin binding by A20 prevents psoriatic arthritis–like disease and inflammation
Authors
Keywords
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Journal
NATURE IMMUNOLOGY
Volume 21, Issue 4, Pages 422-433
Publisher
Springer Science and Business Media LLC
Online
2020-03-17
DOI
10.1038/s41590-020-0634-4
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Note: Only part of the references are listed.- A20: A multifunctional tool for regulating immunity and preventing disease
- (2019) Barbara A. Malynn et al. CELLULAR IMMUNOLOGY
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- (2018) Florence A Aeschlimann et al. ANNALS OF THE RHEUMATIC DISEASES
- Autosomic dominant familial Behçet disease and haploinsufficiency A20: A review of the literature
- (2018) Florian Berteau et al. AUTOIMMUNITY REVIEWS
- Haploinsufficiency of A20 causes autoinflammatory and autoimmune disorders
- (2018) Tomonori Kadowaki et al. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
- The pathogenesis of psoriatic arthritis
- (2018) Douglas J Veale et al. LANCET
- The Immunologic Role of IL-17 in Psoriasis and Psoriatic Arthritis Pathogenesis
- (2018) Andrew Blauvelt et al. CLINICAL REVIEWS IN ALLERGY & IMMUNOLOGY
- Psoriatic Arthritis
- (2017) Christopher T. Ritchlin et al. NEW ENGLAND JOURNAL OF MEDICINE
- Hypomorphic A20 expression confers susceptibility to psoriasis
- (2017) Anri Aki et al. PLoS One
- Recruitment of A20 by the C-terminal domain of NEMO suppresses NF-κB activation and autoinflammatory disease
- (2016) Jevgenia Zilberman-Rudenko et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- A20 Restricts Ubiquitination of Pro-Interleukin-1β Protein Complexes and Suppresses NLRP3 Inflammasome Activity
- (2015) Bao H. Duong et al. IMMUNITY
- Phosphorylation and linear ubiquitin direct A20 inhibition of inflammation
- (2015) Ingrid E. Wertz et al. NATURE
- Loss-of-function mutations in TNFAIP3 leading to A20 haploinsufficiency cause an early-onset autoinflammatory disease
- (2015) Qing Zhou et al. NATURE GENETICS
- The ubiquitin-modifying enzyme A20 restricts ubiquitination of the kinase RIPK3 and protects cells from necroptosis
- (2015) Michio Onizawa et al. NATURE IMMUNOLOGY
- Farm dust and endotoxin protect against allergy through A20 induction in lung epithelial cells
- (2015) M. J. Schuijs et al. SCIENCE
- LUBAC-Recruited CYLD and A20 Regulate Gene Activation and Cell Death by Exerting Opposing Effects on Linear Ubiquitin in Signaling Complexes
- (2015) Peter Draber et al. Cell Reports
- The deubiquitinase activity of A20 is dispensable for NF- B signaling
- (2014) A. De et al. EMBO REPORTS
- Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis
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- Dimerization and Ubiquitin Mediated Recruitment of A20, a Complex Deubiquitinating Enzyme
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- Activation of the canonical IKK complex by K63/M1-linked hybrid ubiquitin chains
- (2013) C. H. Emmerich et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- A20 in inflammation and autoimmunity
- (2013) Leen Catrysse et al. TRENDS IN IMMUNOLOGY
- Expansion of intestinal Prevotella copri correlates with enhanced susceptibility to arthritis
- (2013) Jose U Scher et al. eLife
- A20 inhibits LUBAC-mediated NF-κB activation by binding linear polyubiquitin chains via its zinc finger 7
- (2012) Kelly Verhelst et al. EMBO JOURNAL
- Specific recognition of linear polyubiquitin by A20 zinc finger 7 is involved in NF-κB regulation
- (2012) Fuminori Tokunaga et al. EMBO JOURNAL
- The Ubiquitin-Editing Protein A20 Prevents Dendritic Cell Activation, Recognition of Apoptotic Cells, and Systemic Autoimmunity
- (2011) Mirjam Kool et al. IMMUNITY
- Direct, Noncatalytic Mechanism of IKK Inhibition by A20
- (2011) Brian Skaug et al. MOLECULAR CELL
- A20 (TNFAIP3) deficiency in myeloid cells triggers erosive polyarthritis resembling rheumatoid arthritis
- (2011) Mourad Matmati et al. NATURE GENETICS
- Expression of A20 by dendritic cells preserves immune homeostasis and prevents colitis and spondyloarthritis
- (2011) Gianna Elena Hammer et al. NATURE IMMUNOLOGY
- Gut-Residing Segmented Filamentous Bacteria Drive Autoimmune Arthritis via T Helper 17 Cells
- (2010) Hsin-Jung Wu et al. IMMUNITY
- The Ubiquitin Modifying Enzyme A20 Restricts B Cell Survival and Prevents Autoimmunity
- (2010) Rita M. Tavares et al. IMMUNITY
- Ubiquitin Binding to A20 ZnF4 Is Required for Modulation of NF-κB Signaling
- (2010) Ivan Bosanac et al. MOLECULAR CELL
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- (2010) N. Shembade et al. SCIENCE
- Induction of Intestinal Th17 Cells by Segmented Filamentous Bacteria
- (2009) Ivaylo I. Ivanov et al. CELL
- Structural basis for specific recognition of Lys 63-linked polyubiquitin chains by tandem UIMs of RAP80
- (2009) Yusuke Sato et al. EMBO JOURNAL
- Linkage-Specific Avidity Defines the Lysine 63-Linked Polyubiquitin-Binding Preference of Rap80
- (2009) Joshua J. Sims et al. MOLECULAR CELL
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- (2008) Maria Armaka et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20
- (2008) Emre E. Turer et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Molecular Basis for the Unique Deubiquitinating Activity of the NF-κB Inhibitor A20
- (2007) Su-Chang Lin et al. JOURNAL OF MOLECULAR BIOLOGY
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