High Dietary Advanced Glycation End Products Impair Mitochondrial and Cognitive Function

Anxiety and task performance changes in an aging mouse model

(2019) Erika D. Nolte et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

RAGE mediates Aβ accumulation in a mouse model of Alzheimer’s disease via modulation of β- and γ-secretase activity

(2018) Fang Fang et al.   HUMAN MOLECULAR GENETICS

Overexpression of endophilin A1 exacerbates synaptic alterations in a mouse model of Alzheimer’s disease

(2018) Qing Yu et al.   Nature Communications

Mitochondrial Dysfunction Triggers Synaptic Deficits via Activation of p38 MAP Kinase Signaling in Differentiated Alzheimer’s Disease Trans-Mitochondrial Cybrid Cells

(2017) Qing Yu et al.   JOURNAL OF ALZHEIMERS DISEASE

F 1 F 0 ATP Synthase–Cyclophilin D Interaction Contributes to Diabetes-Induced Synaptic Dysfunction and Cognitive Decline

(2016) Shijun Yan et al.   DIABETES

Pathways of the Maillard reaction under physiological conditions

(2016) Christian Henning et al.   GLYCOCONJUGATE JOURNAL

Increased Electron Paramagnetic Resonance Signal Correlates with Mitochondrial Dysfunction and Oxidative Stress in an Alzheimer’s disease Mouse Brain

(2016) Du Fang et al.   JOURNAL OF ALZHEIMERS DISEASE

Detection of Circulating Auto-Antibodies Against Ribosylated-LDL in Diabetes Patients

(2016) Firoz Akhter et al.   JOURNAL OF CLINICAL LABORATORY ANALYSIS

5-Hydroxy-7-Methoxyflavone Triggers Mitochondrial-Associated Cell Death via Reactive Oxygen Species Signaling in Human Colon Carcinoma Cells

(2016) Monika Bhardwaj et al.   PLoS One

Dicarbonyl stress in cell and tissue dysfunction contributing to ageing and disease

(2015) Naila Rabbani et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

Increased neuronal PreP activity reduces Aβ accumulation, attenuates neuroinflammation and improves mitochondrial and synaptic function in Alzheimer disease's mouse model

(2015) Du Fang et al.   HUMAN MOLECULAR GENETICS

Advanced glycation End-products (AGEs): an emerging concern for processed food industries

(2015) Chetan Sharma et al.   JOURNAL OF FOOD SCIENCE AND TECHNOLOGY-MYSORE

Glycation of H1 Histone by 3-Deoxyglucosone: Effects on Protein Structure and Generation of Different Advanced Glycation End Products

(2015) Jalaluddin Mohammad Ashraf et al.   PLoS One

Drp1-Mediated Mitochondrial Abnormalities Link to Synaptic Injury in Diabetes Model

(2014) Shengbin Huang et al.   DIABETES

Physicochemical analysis of structural alteration and advanced glycation end products generation during glycation of H2A histone by 3-deoxyglucosone

(2014) Jalaluddin M. Ashraf et al.   IUBMB LIFE

Genetic deficiency of neuronal RAGE protects against AGE-induced synaptic injury

(2014) Hongju Zhang et al.   Cell Death & Disease

Advanced glycoxidation and lipoxidation end products (AGEs and ALEs): an overview of their mechanisms of formation

(2013) G. Vistoli et al.   FREE RADICAL RESEARCH

Studies on glycation of human low density lipoprotein: A functional insight into physico-chemical analysis

(2013) Saheem Ahmad et al.   INTERNATIONAL JOURNAL OF BIOLOGICAL MACROMOLECULES

A novel mechanism for antiglycative action of limonene through stabilization of protein conformation

(2013) Madhav M. Joglekar et al.   Molecular BioSystems

Advanced glycation end product level, diabetes, and accelerated cognitive aging

(2011) K. Yaffe et al.   NEUROLOGY

Advanced Glycation End Products in Foods and a Practical Guide to Their Reduction in the Diet

(2010) Jaime Uribarri et al.   JOURNAL OF THE AMERICAN DIETETIC ASSOCIATION

Does Accumulation of Advanced Glycation End Products Contribute to the Aging Phenotype?

(2010) R. D. Semba et al.   JOURNALS OF GERONTOLOGY SERIES A-BIOLOGICAL SCIENCES AND MEDICAL SCIENCES

Early deficits in synaptic mitochondria in an Alzheimer's disease mouse model

(2010) H. Du et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Dietary Advanced Glycation End Products and Aging

(2010) Claudia Luevano-Contreras et al.   Nutrients

RAGE-dependent signaling in microglia contributes to neuroinflammation, Aβ accumulation, and impaired learning/memory in a mouse model of Alzheimer’s disease

(2009) Fang Fang et al.   FASEB JOURNAL

Protection against Loss of Innate Defenses in Adulthood by Low Advanced Glycation End Products (AGE) Intake: Role of the Antiinflammatory AGE Receptor-1

(2009) Helen Vlassara et al.   JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM

Cyclophilin D deficiency improves mitochondrial function and learning/memory in aging Alzheimer disease mouse model

(2009) Heng Du et al.   NEUROBIOLOGY OF AGING

Cyclophilin D deficiency attenuates mitochondrial and neuronal perturbation and ameliorates learning and memory in Alzheimer's disease

(2008) Heng Du et al.   NATURE MEDICINE