Glyceraldehyde-Derived Pyridinium Evokes Renal Tubular Cell Damage via RAGE Interaction
Published 2020 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Glyceraldehyde-Derived Pyridinium Evokes Renal Tubular Cell Damage via RAGE Interaction
Authors
Keywords
-
Journal
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
Volume 21, Issue 7, Pages 2604
Publisher
MDPI AG
Online
2020-04-10
DOI
10.3390/ijms21072604
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Empagliflozin reduces high glucose-induced oxidative stress and miR-21-dependent TRAF3IP2 induction and RECK suppression, and inhibits human renal proximal tubular epithelial cell migration and epithelial-to-mesenchymal transition
- (2019) Nitin A. Das et al. CELLULAR SIGNALLING
- Renoprotection of dapagliflozin in human renal proximal tubular cells via the inhibition of the high mobility group box 1‑receptor for advanced glycation end products‑nuclear factor‑κB signaling pathway
- (2018) Di Yao et al. Molecular Medicine Reports
- Tofogliflozin, a selective inhibitor of sodium-glucose cotransporter 2, suppresses renal damage in KKAy/Ta mice, obese and type 2 diabetic animals
- (2016) Yuji Ishibashi et al. Diabetes & Vascular Disease Research
- Kallistatin protects against diabetic nephropathy in db/db mice by suppressing AGE-RAGE-induced oxidative stress
- (2016) Wai Han Yiu et al. KIDNEY INTERNATIONAL
- Glycation and cardiovascular disease in diabetes: A perspective on the concept of metabolic memory
- (2016) Sho-ichi Yamagishi et al. Journal of Diabetes
- Glyceraldehyde-derived pyridinium (GLAP) evokes oxidative stress and inflammatory and thrombogenic reactions in endothelial cells via the interaction with RAGE
- (2015) Takanori Matsui et al. Cardiovascular Diabetology
- Tofogliflozin, A Highly Selective Inhibitor of SGLT2 Blocks Proinflammatory and Proapoptotic Effects of Glucose Overload on Proximal Tubular Cells Partly by Suppressing Oxidative Stress Generation
- (2015) Y. Ishibashi et al. HORMONE AND METABOLIC RESEARCH
- Empagliflozin, an Inhibitor of Sodium-Glucose Cotransporter 2 Exerts Anti-Inflammatory and Antifibrotic Effects on Experimental Diabetic Nephropathy Partly by Suppressing AGEs-Receptor Axis
- (2015) A. Ojima et al. HORMONE AND METABOLIC RESEARCH
- Unknown
- (2015) Sho-ichi Yamagishi et al. MOLECULAR MEDICINE
- DNA Aptamer Raised Against AGEs Blocks the Progression of Experimental Diabetic Nephropathy
- (2013) Yusuke Kaida et al. DIABETES
- Sodium-glucose cotransporter 2-mediated oxidative stress augments advanced glycation end products-induced tubular cell apoptosis
- (2013) Sayaka Maeda et al. DIABETES-METABOLISM RESEARCH AND REVIEWS
- Positive Association Between Serum Level of Glyceraldehyde-Derived Advanced Glycation End Products and Vascular Inflammation Evaluated by [18F]Fluorodeoxyglucose Positron Emission Tomography
- (2012) N. Tahara et al. DIABETES CARE
- Serum Levels of Advanced Glycation End Products (AGEs) are Independent Correlates of Insulin Resistance in Nondiabetic Subjects
- (2010) Nobuhiro Tahara et al. Cardiovascular Therapeutics
- Irbesartan inhibits advanced glycation end product (AGE)-induced proximal tubular cell injury in vitro by suppressing receptor for AGEs (RAGE) expression
- (2009) Takanori Matsui et al. PHARMACOLOGICAL RESEARCH
- The Formation of Argpyrimidine in Glyceraldehyde-Related Glycation
- (2008) Teruyuki USUI et al. BIOSCIENCE BIOTECHNOLOGY AND BIOCHEMISTRY
Create your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create NowBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started