SCO-Spondin Defects and Neuroinflammation Are Conserved Mechanisms Driving Spinal Deformity across Genetic Models of Idiopathic Scoliosis

Title
SCO-Spondin Defects and Neuroinflammation Are Conserved Mechanisms Driving Spinal Deformity across Genetic Models of Idiopathic Scoliosis
Authors
Keywords
adolescent idiopathic scoliosis, zebrafish, subcommissural organ, SCO-spondin, Reissner’s fiber, cerebrospinal fluid, neuroinflammation, oxidative stress, cyclooxygenase inhibitor, N-acetyl-L-cysteine ethyl ester
Journal
CURRENT BIOLOGY
Volume -, Issue -, Pages -
Publisher
Elsevier BV
Online
2020-05-07
DOI
10.1016/j.cub.2020.04.020

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