Angiotensin-(1–9) prevents cardiomyocyte hypertrophy by controlling mitochondrial dynamics via miR-129-3p/PKIA pathway

Down Syndrome Critical Region 1 Gene,Rcan1, Helps Maintain a More Fused Mitochondrial NetworkNovelty and Significance

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Genome-wide circulating microRNA expression profiling reveals potential biomarkers for amyotrophic lateral sclerosis

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Gene Therapy With Angiotensin-(1-9) Preserves Left Ventricular Systolic Function After Myocardial Infarction

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The Lupus Autoantigen La Prevents Mis-channeling of tRNA Fragments into the Human MicroRNA Pathway

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Mitochondrial fission is required for cardiomyocyte hypertrophy mediated by a Ca2+-calcineurin signaling pathway

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Angiotensin-(1–9) reverses experimental hypertension and cardiovascular damage by inhibition of the angiotensin converting enzyme/Ang II axis

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Insulin Stimulates Mitochondrial Fusion and Function in Cardiomyocytes via the Akt-mTOR-NF B-Opa-1 Signaling Pathway

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Mitochondrial division/mitophagy inhibitor (Mdivi) Ameliorates Pressure Overload Induced Heart Failure

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Angiotensin II type 1 receptor signalling regulates microRNA differentially in cardiac fibroblasts and myocytes

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Mitochondrial Oxidative Stress Mediates Angiotensin II–Induced Cardiac Hypertrophy and Gαq Overexpression–Induced Heart Failure

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Mitochondrial Fusion is Essential for Organelle Function and Cardiac Homeostasis

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Angiotensin-(1-9) Attenuates Cardiac Fibrosis in the Stroke-Prone Spontaneously Hypertensive Rat via the Angiotensin Type 2 Receptor

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Rho kinase inhibition activates the homologous angiotensin-converting enzyme-angiotensin-(1–9) axis in experimental hypertension

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MicroRNA-328 Contributes to Adverse Electrical Remodeling in Atrial Fibrillation

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Inhibiting Mitochondrial Fission Protects the Heart Against Ischemia/Reperfusion Injury

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An Inositol 1,4,5-Triphosphate (IP3)-IP3Receptor Pathway Is Required for Insulin-Stimulated Glucose Transporter 4 Translocation and Glucose Uptake in Cardiomyocytes

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Angiotensin-(1–9) regulates cardiac hypertrophy in vivo and in vitro

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A Mutation in the Mitochondrial Fission Gene Dnm1l Leads to Cardiomyopathy

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Iron induces protection and necrosis in cultured cardiomyocytes: Role of reactive oxygen species and nitric oxide

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MicroRNA Expression Signature and the Role of MicroRNA-21 in the Early Phase of Acute Myocardial Infarction

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Mitochondrial fission mediates high glucose-induced cell death through elevated production of reactive oxygen species

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Does Contractile Ca2+ Control Calcineurin-NFAT Signaling and Pathological Hypertrophy in Cardiac Myocytes?

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