Normalization of cardiac substrate utilization and left ventricular hypertrophy precede functional recovery in heart failure regression

Is AMPK the savior of the failing heart?

(2015) Ty T. Kim et al.   TRENDS IN ENDOCRINOLOGY AND METABOLISM

Energy Metabolic Reprogramming in the Hypertrophied and Early Stage Failing Heart

(2014) Ling Lai et al.   Circulation-Heart Failure

Metformin Protects Against Systolic Overload–Induced Heart Failure Independent of AMP-Activated Protein Kinase α2

(2014) Xin Xu et al.   HYPERTENSION

Cardiac Insulin-Resistance and Decreased Mitochondrial Energy Production Precede the Development of Systolic Heart Failure After Pressure-Overload Hypertrophy

(2013) Liyan Zhang et al.   Circulation-Heart Failure

Pressure-overload-induced heart failure induces a selective reduction in glucose oxidation at physiological afterload

(2012) Pavel Zhabyeyev et al.   CARDIOVASCULAR RESEARCH

Unraveling new mechanisms of exercise intolerance in chronic heart failure. Role of exercise training

(2012) Viviane M. Conraads et al.   HEART FAILURE REVIEWS

Recovery from decompensated heart failure is associated with a distinct, phase-dependent gene expression profile

(2012) Nancy M. Andersen et al.   JOURNAL OF SURGICAL RESEARCH

AMPK signalling and the control of substrate use in the heart

(2012) Jeevan Nagendran et al.   MOLECULAR AND CELLULAR ENDOCRINOLOGY

Insights into the regulation of protein abundance from proteomic and transcriptomic analyses

(2012) Christine Vogel et al.   NATURE REVIEWS GENETICS

A mouse model of reverse cardiac remodelling following banding-debanding of the ascending aorta

(2011) J. L. Bjørnstad et al.   Acta Physiologica

Calorie restriction and resveratrol in cardiovascular health and disease

(2011) Vernon W. Dolinsky et al.   BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE

Glucose metabolism and cardiac hypertrophy

(2011) S. C. Kolwicz et al.   CARDIOVASCULAR RESEARCH

Cardiac Raptor Ablation Impairs Adaptive Hypertrophy, Alters Metabolic Gene Expression, and Causes Heart Failure in Mice

(2011) Pankaj Shende et al.   CIRCULATION

PGC-1β Deficiency Accelerates the Transition to Heart Failure in Pressure Overload Hypertrophy

(2011) Christian Riehle et al.   CIRCULATION RESEARCH

AKT signalling in the failing heart

(2011) Antoine H. Chaanine et al.   EUROPEAN JOURNAL OF HEART FAILURE

Increased CD36 expression in middle-aged mice contributes to obesity-related cardiac hypertrophy in the absence of cardiac dysfunction

(2011) Miranda M. Y. Sung et al.   JOURNAL OF MOLECULAR MEDICINE-JMM

Myocardial ATGL Overexpression Decreases the Reliance on Fatty Acid Oxidation and Protects against Pressure Overload-Induced Cardiac Dysfunction

(2011) P. C. Kienesberger et al.   MOLECULAR AND CELLULAR BIOLOGY

Angiotensin-Converting Enzyme 2 Suppresses Pathological Hypertrophy, Myocardial Fibrosis, and Cardiac Dysfunction

(2010) JiuChang Zhong et al.   CIRCULATION

Molecular Pathways Underlying Cardiac Remodeling During Pathophysiological Stimulation

(2010) Izhak Kehat et al.   CIRCULATION

Alterations in Skeletal Muscle Fatty Acid Handling Predisposes Middle-Aged Mice to Diet-Induced Insulin Resistance

(2010) D. P. Y. Koonen et al.   DIABETES

Downregulation of survival signalling pathways and increased apoptosis in the transition of pressure overload-induced cardiac hypertrophy to heart failure

(2009) Xiao-Mei Li et al.   CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY

Regression of pressure-induced left ventricular hypertrophy is characterized by a distinct gene expression profile

(2009) William E. Stansfield et al.   JOURNAL OF THORACIC AND CARDIOVASCULAR SURGERY

Global signatures of protein and mRNA expression levels

(2009) Raquel de Sousa Abreu et al.   Molecular BioSystems

The Athleteʼs Heart

(2009) Louise H Naylor et al.   SPORTS MEDICINE

How is mRNA expression predictive for protein expression? A correlation study on human circulating monocytes

(2008) Yanfang Guo et al.   ACTA BIOCHIMICA ET BIOPHYSICA SINICA

Reverse changes in cardiac substrate oxidation in dogs recovering from heart failure

(2008) Khaled Qanud et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY