Hepatitis E Virus Induces Brain Injury Probably Associated With Mitochondrial Apoptosis

Brain Infection by Hepatitis E Virus Probably via Damage of the Blood-Brain Barrier Due to Alterations of Tight Junction Proteins

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Transmission of a Novel Genotype of Hepatitis E Virus from Bactrian Camels to Cynomolgus Macaques

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Berberine protects acute liver failure in mice through inhibiting inflammation and mitochondria-dependent apoptosis

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BAK/BAX macropores facilitate mitochondrial herniation and mtDNA efflux during apoptosis

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Hepatitis E Virus Induces Hepatocyte Apoptosis via Mitochondrial Pathway in Mongolian Gerbils

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Mitochondrial-Directed Antioxidant Reduces Microglial-Induced Inflammation in Murine In Vitro Model of TC-83 Infection

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Hepatitis E Virus: Animal Models and Zoonosis

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Role of Substance P Neuropeptide in Inflammation, Wound Healing, and Tissue Homeostasis

(2017) Susmit Suvas   JOURNAL OF IMMUNOLOGY

Molecular and structural changes related to hepatitis E virus antigen and its expression in testis inducing apoptosis in Mongolian gerbil model

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The Therapeutic Potential of Targeting Substance P/NK-1R Interactions in Inflammatory CNS Disorders

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Hepatitis E virus infection

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A complex V ATP5A1 defect causes fatal neonatal mitochondrial encephalopathy

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Mitochondrial dysfunctions in Myalgic Encephalomyelitis / chronic fatigue syndrome explained by activated immuno-inflammatory, oxidative and nitrosative stress pathways

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Bovine ephemeral fever virus-induced apoptosis requires virus gene expression and activation of Fas and mitochondrial signaling pathway

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Mitochondrial DNA damage is involved in apoptosis caused by pro-inflammatory cytokines in human OA chondrocytes

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