Copy number gains of the putative CRKL oncogene in laryngeal squamous cell carcinoma result in strong nuclear expression of the protein and influence cell proliferation and migration
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Title
Copy number gains of the putative CRKL oncogene in laryngeal squamous cell carcinoma result in strong nuclear expression of the protein and influence cell proliferation and migration
Authors
Keywords
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Journal
Scientific Reports
Volume 10, Issue 1, Pages -
Publisher
Springer Science and Business Media LLC
Online
2020-01-08
DOI
10.1038/s41598-019-56870-5
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- FDA Approval Summary: Pembrolizumab for the Treatment of Recurrent or Metastatic Head and Neck Squamous Cell Carcinoma with Disease Progression on or After Platinum‐Containing Chemotherapy
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- Recurrent amplification in the 22q11 region in laryngeal squamous cell carcinoma results in overexpression of the CRKL but not the MAPK1 oncogene
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- Efficacy and toxicity of cetuximab with chemotherapy in recurrent and metastatic head and neck cancer: A prospective observational study
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- Oncogenic Signaling Adaptor Proteins
- (2015) Leo Y. Luo et al. Journal of Genetics and Genomics
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- (2014) Kenichi Suda et al. LUNG CANCER
- CRKL protein overexpression enhances cell proliferation and invasion in pancreatic cancer
- (2014) Lin Fu et al. TUMOR BIOLOGY
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- Overexpression of CRKL correlates with malignant cell proliferation in breast cancer
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- The CRKL gene encoding an adaptor protein is amplified, overexpressed, and a possible therapeutic target in gastric cancer
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- Estimates of worldwide burden of cancer in 2008: GLOBOCAN 2008
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- Regulation of growth factor signaling by FRS2 family docking/scaffold adaptor proteins
- (2008) Noriko Gotoh CANCER SCIENCE
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