MicroRNA‐129‐1‐3p protects cardiomyocytes from pirarubicin‐induced apoptosis by down‐regulating the GRIN2D‐mediated Ca 2+ signalling pathway
Published 2020 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
MicroRNA‐129‐1‐3p protects cardiomyocytes from pirarubicin‐induced apoptosis by down‐regulating the GRIN2D‐mediated Ca
2+
signalling pathway
Authors
Keywords
-
Journal
JOURNAL OF CELLULAR AND MOLECULAR MEDICINE
Volume 24, Issue 3, Pages 2260-2271
Publisher
Wiley
Online
2020-01-20
DOI
10.1111/jcmm.14908
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Matrine attenuates oxidative stress and cardiomyocyte apoptosis in doxorubicin-induced cardiotoxicity via maintaining AMPKα/UCP2 pathway
- (2019) Can Hu et al. Acta Pharmaceutica Sinica B
- The Cardiotoxicity Induced by Arsenic Trioxide is Alleviated by Salvianolic Acid A via Maintaining Calcium Homeostasis and Inhibiting Endoplasmic Reticulum Stress
- (2019) Ruiying Wang et al. MOLECULES
- Low concentration of rutin treatment might alleviate the cardiotoxicity effect of pirarubicin on cardiomyocytes via activation of PI3K/AKT/mTOR signaling pathway
- (2019) Junjie Fei et al. BIOSCIENCE REPORTS
- Cardiotoxicity and pro-inflammatory effects of the immune checkpoint inhibitor Pembrolizumab associated to Trastuzumab
- (2019) V. Quagliariello et al. INTERNATIONAL JOURNAL OF CARDIOLOGY
- Gene Therapy for Catecholaminergic Polymorphic Ventricular Tachycardia by Inhibition of Ca 2+ /Calmodulin-Dependent Kinase II
- (2019) Vassilios J. Bezzerides et al. CIRCULATION
- Composite Conformational Changes of Signaling Proteins upon Ligand Binding Revealed by a Single Approach: Calcium-Calmodulin Study
- (2019) Xiaoran Roger Liu et al. ANALYTICAL CHEMISTRY
- CaMKII-δ9 promotes cardiomyopathy through disrupting UBE2T-dependent DNA repair
- (2019) Mao Zhang et al. NATURE CELL BIOLOGY
- Efficacy of Flecainide in Catecholaminergic Polymorphic Ventricular Tachycardia Is Mutation-Independent but Reduced by Calcium Overload
- (2019) Hyun Seok Hwang et al. Frontiers in Physiology
- Genetic Mosaicism in Calmodulinopathy
- (2019) Lisa M. Wren et al. Circulation-Genomic and Precision Medicine
- NR1 and NR3B Composed Intranuclear N-methyl-d-aspartate Receptor Complexes in Human Melanoma Cells
- (2018) Tibor Hajdú et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- The cardiac ryanodine receptor, but not sarcoplasmic reticulum Ca2+-ATPase, is a major determinant of Ca2+ alternans in intact mouse hearts
- (2018) Bo Sun et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- MicroRNAs in the diagnosis and prevention of drug-induced cardiotoxicity
- (2018) Mikuláš Skála et al. ARCHIVES OF TOXICOLOGY
- Role of microRNAs in doxorubicin-induced cardiotoxicity: an overview of preclinical models and cancer patients
- (2017) Clarissa Ruggeri et al. HEART FAILURE REVIEWS
- Autophagic dysregulation in doxorubicin cardiomyopathy
- (2017) Jordan J. Bartlett et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Mitochondrial energetics and calcium coupling in the heart
- (2017) Michael Kohlhaas et al. JOURNAL OF PHYSIOLOGY-LONDON
- Myocardin and Stat3 act synergistically to inhibit cardiomyocyte apoptosis
- (2017) Yuan Xiang et al. Oncotarget
- New signal transduction paradigms in anthracycline-induced cardiotoxicity
- (2016) Alessandra Ghigo et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH
- SIRT1 activation attenuates diastolic dysfunction by reducing cardiac fibrosis in a model of anthracycline cardiomyopathy
- (2016) Donato Cappetta et al. INTERNATIONAL JOURNAL OF CARDIOLOGY
- Cardioprotective effects of baicalein on heart failure via modulation of Ca2+ handling proteins in vivo and in vitro
- (2016) Fali Zhao et al. LIFE SCIENCES
- Cardiotoxicidade na terapêutica com antraciclinas: estratégias de prevenção
- (2016) Margarida Cruz et al. Revista Portuguesa de Cardiologia
- CHOP or THP-COP regimens in the treatment of newly diagnosed peripheral T-cell lymphoma, not otherwise specified: a comparison of doxorubicin and pirarubicin
- (2015) Yuhei Shibata et al. HEMATOLOGICAL ONCOLOGY
- Constitutive activation of STAT3 in breast cancer cells: A review
- (2015) Kasturi Banerjee et al. INTERNATIONAL JOURNAL OF CANCER
- MiR-21 Protected Cardiomyocytes against Doxorubicin-Induced Apoptosis by Targeting BTG2
- (2015) Zhongyi Tong et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- MicroRNA-208a Silencing Attenuates Doxorubicin Induced Myocyte Apoptosis and Cardiac Dysfunction
- (2015) Hasahya Tony et al. Oxidative Medicine and Cellular Longevity
- Cardiotoxicity: Anthracyclines and long term cancer survivors
- (2014) Salvatore Patanè INTERNATIONAL JOURNAL OF CARDIOLOGY
- Blood microRNA profile associates with the levels of serum lipids and metabolites associated with glucose metabolism and insulin resistance and pinpoints pathways underlying metabolic syndrome
- (2014) Emma Raitoharju et al. MOLECULAR AND CELLULAR ENDOCRINOLOGY
- Anthracycline-induced cardiotoxicity: Overview of studies examining the roles of oxidative stress and free cellular iron
- (2014) Tomáš Šimůnek et al. Pharmacological Reports
- The Emerging Role of Small Non-coding RNAs in the Failing Heart: Big Hopes for Small Molecules
- (2011) Douglas L. Mann CARDIOVASCULAR DRUGS AND THERAPY
- Targeting the Mitochondrial Permeability Transition: Cardiac Ischemia-Reperfusion Versus Carcinogenesis
- (2011) Sabzali Javadov et al. CELLULAR PHYSIOLOGY AND BIOCHEMISTRY
- Metabonomic study on the cumulative cardiotoxicity of a pirarubicin liposome powder
- (2011) Wenjuan Cong et al. TALANTA
- Respiratory chain deficiency precedes the disrupted calcium homeostasis in chronic doxorubicin cardiomyopathy
- (2009) Dirk Lebrecht et al. CARDIOVASCULAR PATHOLOGY
- MicroRNA and cardiac pathologies
- (2008) Michael V. G. Latronico et al. PHYSIOLOGICAL GENOMICS
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started