Endothelial cell-specific redox gene modulation inhibits angiogenesis but promotes B16F0 tumor growth in mice
Published 2019 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Endothelial cell-specific redox gene modulation inhibits angiogenesis but promotes B16F0 tumor growth in mice
Authors
Keywords
-
Journal
FASEB JOURNAL
Volume 33, Issue 12, Pages 14147-14158
Publisher
FASEB
Online
2019-10-25
DOI
10.1096/fj.201900786r
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Protein S -glutathionylation induced by hypoxia increases hypoxia-inducible factor-1α in human colon cancer cells
- (2018) Daun Jeon et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- SearchGUI: A Highly Adaptable Common Interface for Proteomics Search and de Novo Engines
- (2018) Harald Barsnes et al. JOURNAL OF PROTEOME RESEARCH
- Connective tissue growth factor (CTGF) from basics to clinics
- (2018) Yasaman Ramazani et al. MATRIX BIOLOGY
- Reducing protein oxidation reverses lung fibrosis
- (2018) Vikas Anathy et al. NATURE MEDICINE
- Vasculogenic mimicry signaling revisited: focus on non-vascular VE-cadherin
- (2017) Daniel Delgado-Bellido et al. Molecular Cancer
- Redox regulation of ischemic limb neovascularization – What we have learned from animal studies
- (2017) Reiko Matsui et al. Redox Biology
- Upregulation of Glutaredoxin-1 Activates Microglia and Promotes Neurodegeneration: Implications for Parkinson's Disease
- (2016) Olga Gorelenkova Miller et al. ANTIOXIDANTS & REDOX SIGNALING
- Tumor cell vascular mimicry: Novel targeting opportunity in melanoma
- (2016) Mary J.C. Hendrix et al. PHARMACOLOGY & THERAPEUTICS
- Glutathione adducts induced by ischemia and deletion of glutaredoxin-1 stabilize HIF-1α and improve limb revascularization
- (2016) Yosuke Watanabe et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- The redox mechanism for vascular barrier dysfunction associated with metabolic disorders: Glutathionylation of Rac1 in endothelial cells
- (2016) Jingyan Han et al. Redox Biology
- Induction of Vasculogenic Mimicry Overrides VEGF-A Silencing and Enriches Stem-like Cancer Cells in Melanoma
- (2015) C. I. Schnegg et al. CANCER RESEARCH
- PeptideShaker enables reanalysis of MS-derived proteomics data sets
- (2015) Marc Vaudel et al. NATURE BIOTECHNOLOGY
- Integrated analysis of shotgun proteomic data with PatternLab for proteomics 4.0
- (2015) Paulo C Carvalho et al. Nature Protocols
- Plasminogen Activator Inhibitor-1 Controls Vascular Integrity by Regulating VE-Cadherin Trafficking
- (2015) Anna E. Daniel et al. PLoS One
- Glutaredoxin-1 AttenuatesS-Glutathionylation of the Death Receptor Fas and Decreases Resolution ofPseudomonas aeruginosaPneumonia
- (2014) Vikas Anathy et al. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE
- ROS Function in Redox Signaling and Oxidative Stress
- (2014) Michael Schieber et al. CURRENT BIOLOGY
- Does reversible cysteine oxidation link the Western diet to cardiac dysfunction?
- (2014) Jessica B. Behring et al. FASEB JOURNAL
- Glutaredoxin-1 Up-regulation Induces Soluble Vascular Endothelial Growth Factor Receptor 1, Attenuating Post-ischemia Limb Revascularization
- (2014) Colin E. Murdoch et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Vascular channels formed by subpopulations of PECAM1+ melanoma cells
- (2014) James M. Dunleavey et al. Nature Communications
- Redox Regulation of SERCA2 Is Required for Vascular Endothelial Growth Factor-Induced Signaling and Endothelial Cell Migration
- (2012) Alicia M. Evangelista et al. ANTIOXIDANTS & REDOX SIGNALING
- Hypoxia Negatively Regulates Antimetastatic PEDF in Melanoma Cells by a Hypoxia Inducible Factor-Independent, Autophagy Dependent Mechanism
- (2012) Asunción Fernández-Barral et al. PLoS One
- Vasculogenic Mimicry of HT1080 Tumour Cells In Vivo: Critical Role of HIF-1α-Neuropilin-1 Axis
- (2012) Roli M. Misra et al. PLoS One
- Antiangiogenic agents increase breast cancer stem cells via the generation of tumor hypoxia
- (2012) S. J. Conley et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Regulation of Cell Physiology and Pathology by Protein S-Glutathionylation: Lessons Learned from the Cardiovascular System
- (2011) David Pimentel et al. ANTIOXIDANTS & REDOX SIGNALING
- Role of Twist in vasculogenic mimicry formation in hypoxic hepatocellular carcinoma cells in vitro
- (2011) Ma Jin-lu et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- VEGFR-1 Expressed by Malignant Melanoma-Initiating Cells Is Required for Tumor Growth
- (2011) Natasha Y. Frank et al. CANCER RESEARCH
- Inhibition of Pyruvate Kinase M2 by Reactive Oxygen Species Contributes to Cellular Antioxidant Responses
- (2011) D. Anastasiou et al. SCIENCE
- Functional significance of VEGF-a in human ovarian carcinoma: Role in vasculogenic mimicry
- (2010) Jun-yan Wang et al. CANCER BIOLOGY & THERAPY
- Attenuated cardiovascular hypertrophy and oxidant generation in response to angiotensin II infusion in glutaredoxin-1 knockout mice
- (2010) Markus M. Bachschmid et al. FREE RADICAL BIOLOGY AND MEDICINE
- Glioblastoma stem-like cells give rise to tumour endothelium
- (2010) Rong Wang et al. NATURE
- Redox amplification of apoptosis by caspase-dependent cleavage of glutaredoxin 1 and S-glutathionylation of Fas
- (2009) Vikas Anathy et al. JOURNAL OF CELL BIOLOGY
- Evaluation of postnatal arteriogenesis and angiogenesis in a mouse model of hind-limb ischemia
- (2009) Anne Limbourg et al. Nature Protocols
- Protein S-glutathionylation: a regulatory device from bacteria to humans
- (2009) Isabella Dalle-Donne et al. TRENDS IN BIOCHEMICAL SCIENCES
- Redox-based regulation of signal transduction: Principles, pitfalls, and promises
- (2008) Yvonne M.W. Janssen-Heininger et al. FREE RADICAL BIOLOGY AND MEDICINE
Find the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
SearchAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started