Cardiac sympathetic nerve transdifferentiation reduces action potential heterogeneity after myocardial infarction
Published 2020 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Cardiac sympathetic nerve transdifferentiation reduces action potential heterogeneity after myocardial infarction
Authors
Keywords
-
Journal
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
Volume 318, Issue 3, Pages H558-H565
Publisher
American Physiological Society
Online
2020-01-24
DOI
10.1152/ajpheart.00412.2019
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- β-Adrenergic Receptor Stimulation and Alternans in the Border Zone of a Healed Infarct: An ex vivo Study and Computational Investigation of Arrhythmogenesis
- (2019) Jakub Tomek et al. Frontiers in Physiology
- Different paths, same destination: divergent action potential responses produce conserved cardiac fight‐or‐flight response in mouse and rabbit hearts
- (2019) Lianguo Wang et al. JOURNAL OF PHYSIOLOGY-LONDON
- Guidelines for experimental models of myocardial ischemia and infarction
- (2018) Merry L. Lindsey et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Age-related changes in cardiac electrophysiology and calcium handling in response to sympathetic nerve stimulation
- (2018) Samantha D. Francis Stuart et al. JOURNAL OF PHYSIOLOGY-LONDON
- Transcriptomic and neurochemical analysis of the stellate ganglia in mice highlights sex differences
- (2018) R. G. Bayles et al. Scientific Reports
- Molecular Mechanisms of Sympathetic Remodeling and Arrhythmias
- (2016) Ryan T. Gardner et al. Circulation-Arrhythmia and Electrophysiology
- Myocardial Infarction Causes Transient Cholinergic Transdifferentiation of Cardiac Sympathetic Nerves via gp130
- (2016) A. Olivas et al. JOURNAL OF NEUROSCIENCE
- Atherosclerosis exacerbates arrhythmia following myocardial infarction: Role of myocardial inflammation
- (2015) Nicole M. De Jesus et al. HEART RHYTHM
- Myocardial infarction induces structural and functional remodelling of the intrinsic cardiac nervous system
- (2015) Pradeep S. Rajendran et al. JOURNAL OF PHYSIOLOGY-LONDON
- Targeting protein tyrosine phosphatase σ after myocardial infarction restores cardiac sympathetic innervation and prevents arrhythmias
- (2015) R. T. Gardner et al. Nature Communications
- Na+-induced Ca2+ influx through reverse mode of Na+-Ca2+ exchanger in mouse ventricular cardiomyocyte
- (2015) Zhen-Yu Yan et al. Oncotarget
- Optical Mapping of Sarcoplasmic Reticulum Ca 2+ in the Intact Heart
- (2014) Lianguo Wang et al. CIRCULATION RESEARCH
- Altered norepinephrine content and ventricular function in p75NTR−/− mice after myocardial infarction
- (2011) Christina U. Lorentz et al. AUTONOMIC NEUROSCIENCE-BASIC & CLINICAL
- Generation of the tamoxifen-inducible DBH-Cre transgenic mouse line DBH-CT
- (2011) Jutta Stubbusch et al. GENESIS
- Microvolt T-Wave Alternans
- (2011) Richard L. Verrier et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- Heart failure causes cholinergic transdifferentiation of cardiac sympathetic nerves via gp130-signaling cytokines in rodents
- (2010) Hideaki Kanazawa et al. JOURNAL OF CLINICAL INVESTIGATION
- Alternans of action potential duration and amplitude in rabbits with left ventricular dysfunction following myocardial infarction
- (2010) Rachel C. Myles et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Cardiac ischemia-reperfusion regulates sympathetic neuropeptide expression through gp130-dependent and independent mechanisms
- (2010) Eric N. Alston et al. NEUROPEPTIDES
- Infarction-induced cytokines cause local depletion of tyrosine hydroxylase in cardiac sympathetic nerves
- (2009) Diana C. Parrish et al. EXPERIMENTAL PHYSIOLOGY
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationPublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More