Imbalance in the response of pre- and post-synaptic components to amyloidopathy
Published 2019 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Imbalance in the response of pre- and post-synaptic components to amyloidopathy
Authors
Keywords
-
Journal
Scientific Reports
Volume 9, Issue 1, Pages -
Publisher
Springer Science and Business Media LLC
Online
2019-10-16
DOI
10.1038/s41598-019-50781-1
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- APOE genotype and sex affect microglial interactions with plaques in Alzheimer’s disease mice
- (2019) T. L. Stephen et al. Acta Neuropathologica Communications
- Targeting the Synapse in Alzheimer’s Disease
- (2019) Johanna Jackson et al. Frontiers in Neuroscience
- Wnt Signaling Mediates LTP-Dependent Spine Plasticity and AMPAR Localization through Frizzled-7 Receptors
- (2018) Faye McLeod et al. Cell Reports
- The increasing impact of cerebral amyloid angiopathy: essential new insights for clinical practice
- (2017) Gargi Banerjee et al. JOURNAL OF NEUROLOGY NEUROSURGERY AND PSYCHIATRY
- Identification of a peptide recognizing cerebrovascular changes in mouse models of Alzheimer’s disease
- (2017) Aman P. Mann et al. Nature Communications
- Altered Synapse Stability in the Early Stages of Tauopathy
- (2017) Johanna S. Jackson et al. Cell Reports
- APP, PSEN1, and PSEN2 mutations in early-onset Alzheimer disease: A genetic screening study of familial and sporadic cases
- (2017) Hélène-Marie Lanoiselée et al. PLOS MEDICINE
- TREM2-mediated early microglial response limits diffusion and toxicity of amyloid plaques
- (2016) Yaming Wang et al. JOURNAL OF EXPERIMENTAL MEDICINE
- TREM2 Haplodeficiency in Mice and Humans Impairs the Microglia Barrier Function Leading to Decreased Amyloid Compaction and Severe Axonal Dystrophy
- (2016) Peng Yuan et al. NEURON
- Complement and microglia mediate early synapse loss in Alzheimer mouse models
- (2016) S. Hong et al. SCIENCE
- Amyloid Plaques in Retina for Diagnosis in Alzheimer’s Patients: a Meta-Analysis
- (2016) Jiangling Jiang et al. Frontiers in Aging Neuroscience
- Behavioural Phenotyping of APPswe/PS1δE9 Mice: Age-Rrelated Changes and Effect of Long-Term Paroxetine Treatment
- (2016) Louise Ørum Olesen et al. PLoS One
- Network Disruption and Cerebrospinal Fluid Amyloid-Beta and Phospho-Tau Levels in Mild Cognitive Impairment
- (2015) L. Canuet et al. JOURNAL OF NEUROSCIENCE
- Microglia constitute a barrier that prevents neurotoxic protofibrillar Aβ42 hotspots around plaques
- (2015) Carlo Condello et al. Nature Communications
- In vivo imaging reveals sigmoidal growth kinetic of β-amyloid plaques
- (2014) Steffen Burgold et al. Acta Neuropathologica Communications
- Clustering of plaques contributes to plaque growth in a mouse model of Alzheimer’s disease
- (2013) Joanna F. McCarter et al. ACTA NEUROPATHOLOGICA
- Dendritic Spine Instability Leads to Progressive Neocortical Spine Loss in a Mouse Model of Huntington's Disease
- (2013) R. P. Murmu et al. JOURNAL OF NEUROSCIENCE
- Chronic γ-secretase inhibition reduces amyloid plaque-associated instability of pre- and postsynaptic structures
- (2013) S Liebscher et al. MOLECULAR PSYCHIATRY
- Increased axonal bouton dynamics in the aging mouse cortex
- (2013) F. W. Grillo et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Human LilrB2 Is a -Amyloid Receptor and Its Murine Homolog PirB Regulates Synaptic Plasticity in an Alzheimer's Model
- (2013) T. Kim et al. SCIENCE
- A New Player in the “Synaptopathy” of Alzheimer’s Disease – Arc/Arg 3.1
- (2013) Talitha L. Kerrigan et al. Frontiers in Neurology
- A Plaque-Specific Antibody Clears Existing β-amyloid Plaques in Alzheimer's Disease Mice
- (2012) Ronald B. DeMattos et al. NEURON
- Long-Term In Vivo Imaging of -Amyloid Plaque Appearance and Growth in a Mouse Model of Cerebral -Amyloidosis
- (2011) J. K. Hefendehl et al. JOURNAL OF NEUROSCIENCE
- Altered intrinsic neuronal excitability and reduced Na+ currents in a mouse model of Alzheimer's disease
- (2011) Jon T. Brown et al. NEUROBIOLOGY OF AGING
- Multicolor time-stamp reveals the dynamics and toxicity of amyloid deposition
- (2011) Carlo Condello et al. Scientific Reports
- Delayed Stabilization of Dendritic Spines in Fragile X Mice
- (2010) A. Cruz-Martin et al. JOURNAL OF NEUROSCIENCE
- Tau Mislocalization to Dendritic Spines Mediates Synaptic Dysfunction Independently of Neurodegeneration
- (2010) Brian R. Hoover et al. NEURON
- The functional neurophysiology of the amyloid precursor protein (APP) processing pathway
- (2010) A.D. Randall et al. NEUROPHARMACOLOGY
- Multiple Events Lead to Dendritic Spine Loss in Triple Transgenic Alzheimer's Disease Mice
- (2010) Tobias Bittner et al. PLoS One
- Scavenger receptor class B type I (SR-BI) regulates perivascular macrophages and modifies amyloid pathology in an Alzheimer mouse model
- (2010) K. Thanopoulou et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- The p75 Neurotrophin Receptor Promotes Amyloid- (1-42)-Induced Neuritic Dystrophy In Vitro and In Vivo
- (2009) J. K. Knowles et al. JOURNAL OF NEUROSCIENCE
- Hypothetical model of dynamic biomarkers of the Alzheimer's pathological cascade
- (2009) Clifford R Jack et al. LANCET NEUROLOGY
- Oligomeric amyloid associates with postsynaptic densities and correlates with excitatory synapse loss near senile plaques
- (2009) R. M. Koffie et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Rapid appearance and local toxicity of amyloid-β plaques in a mouse model of Alzheimer’s disease
- (2008) Melanie Meyer-Luehmann et al. NATURE
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started