OPA1 overexpression ameliorates mitochondrial cristae remodeling, mitochondrial dysfunction, and neuronal apoptosis in prion diseases
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Title
OPA1 overexpression ameliorates mitochondrial cristae remodeling, mitochondrial dysfunction, and neuronal apoptosis in prion diseases
Authors
Keywords
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Journal
Cell Death & Disease
Volume 10, Issue 10, Pages -
Publisher
Springer Science and Business Media LLC
Online
2019-10-16
DOI
10.1038/s41419-019-1953-y
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- DLP1-dependent mitochondrial fragmentation mediates 1-methyl-4-phenylpyridinium toxicity in neurons: implications for Parkinson’s disease
- (2011) Xinglong Wang et al. AGING CELL
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- (2011) Ligia C. Gomes et al. CELL CYCLE
- Abnormal mitochondrial dynamics, mitochondrial loss and mutant huntingtin oligomers in Huntington's disease: implications for selective neuronal damage
- (2011) U. Shirendeb et al. HUMAN MOLECULAR GENETICS
- Dynamic regulation of mitochondrial fission through modification of the dynamin-related protein Drp1
- (2010) Chuang-Rung Chang et al. Annals of the New York Academy of Sciences
- Mitochondrial Fusion Is Required for mtDNA Stability in Skeletal Muscle and Tolerance of mtDNA Mutations
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- Mitochondrial fission and cristae disruption increase the response of cell models of Huntington's disease to apoptotic stimuli
- (2010) Veronica Costa et al. EMBO Molecular Medicine
- OPA1 links human mitochondrial genome maintenance to mtDNA replication and distribution
- (2010) G. Elachouri et al. GENOME RESEARCH
- Mff is an essential factor for mitochondrial recruitment of Drp1 during mitochondrial fission in mammalian cells
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- (2008) Gilad Twig et al. EMBO JOURNAL
- Opa1-Mediated Cristae Opening Is Bax/Bak and BH3 Dependent, Required for Apoptosis, and Independent of Bak Oligomerization
- (2008) Ryuji Yamaguchi et al. MOLECULAR CELL
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