Endothelial Cell-Derived IL-18 Released During Ischemia Reperfusion Injury Selectively Expands T Peripheral Helper Cells to Promote Alloantibody Production
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Title
Endothelial Cell-Derived IL-18 Released During Ischemia Reperfusion Injury Selectively Expands T Peripheral Helper Cells to Promote Alloantibody Production
Authors
Keywords
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Journal
CIRCULATION
Volume -, Issue -, Pages -
Publisher
Ovid Technologies (Wolters Kluwer Health)
Online
2019-11-20
DOI
10.1161/circulationaha.119.042501
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- The XIIIth Banff Conference on Allograft Pathology: The Banff 2015 Heart Meeting Report: Improving Antibody-Mediated Rejection Diagnostics: Strengths, Unmet Needs, and Future Directions
- (2016) P. Bruneval et al. AMERICAN JOURNAL OF TRANSPLANTATION
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- (2014) L. G. Barnett et al. JOURNAL OF IMMUNOLOGY
- Renal Ischemia-Reperfusion Injury Amplifies the Humoral Immune Response
- (2013) R. Fuquay et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
- Complement-Binding Anti-HLA Antibodies and Kidney-Allograft Survival
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- Serum Neutrophil Gelatinase-Associated Lipocalin and Interleukin-18 as Predictive Biomarkers for Delayed Graft Function After Kidney Transplantation
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- (2009) R. J. Johnston et al. SCIENCE
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