TRIM27 promotes the development of esophagus cancer via regulating PTEN/AKT signaling pathway
Published 2019 View Full Article
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Title
TRIM27 promotes the development of esophagus cancer via regulating PTEN/AKT signaling pathway
Authors
Keywords
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Journal
Cancer Cell International
Volume 19, Issue 1, Pages -
Publisher
Springer Science and Business Media LLC
Online
2019-11-08
DOI
10.1186/s12935-019-0998-4
References
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Related references
Note: Only part of the references are listed.- Endoscopic Management of Early Adenocarcinoma and Squamous Cell Carcinoma of the Esophagus: Screening, Diagnosis, and Therapy
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- (2017) Shigetsugu Hatakeyama TRENDS IN BIOCHEMICAL SCIENCES
- TRIM14 regulates cell proliferation and invasion in osteosarcoma via promotion of the AKT signaling pathway
- (2017) Guoxing Xu et al. Scientific Reports
- Cancer statistics for Hispanics/Latinos, 2015
- (2015) Rebecca L. Siegel et al. CA-A CANCER JOURNAL FOR CLINICIANS
- Downregulation of TRIM27 expression inhibits the proliferation of ovarian cancer cells in vitro and in vivo
- (2015) Yanyan Ma et al. LABORATORY INVESTIGATION
- PI3K/AKT signaling pathway and cancer: an updated review
- (2014) Miriam Martini et al. ANNALS OF MEDICINE
- Baicalein reverses hypoxia-induced 5-FU resistance in gastric cancer AGS cells through suppression of glycolysis and the PTEN/Akt/HIF-1α signaling pathway
- (2014) FENGLIN CHEN et al. ONCOLOGY REPORTS
- Variations in the MHC Region Confer Risk to Esophageal Squamous Cell Carcinoma on the Subjects from High-Incidence Area in Northern China
- (2014) Fang-Fang Shen et al. PLoS One
- RFP-mediated ubiquitination of PTEN modulates its effect on AKT activation
- (2013) James T Lee et al. CELL RESEARCH
- Ubiquitination-Deubiquitination by the TRIM27-USP7 Complex Regulates Tumor Necrosis Factor Alpha-Induced Apoptosis
- (2013) M. M.-U. Zaman et al. MOLECULAR AND CELLULAR BIOLOGY
- Tumor glycolysis as a target for cancer therapy: progress and prospects
- (2013) Shanmugasundaram Ganapathy-Kanniappan et al. Molecular Cancer
- Loss of function of PTEN alters the relationship between glucose concentration and cell proliferation, increases glycolysis, and sensitizes cells to 2-deoxyglucose
- (2009) Marie-José Blouin et al. CANCER LETTERS
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