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Title
SLC4A11 Function: Evidence for H+(OH-) and NH3-H+ Transport
Authors
Keywords
-
Journal
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
Volume -, Issue -, Pages -
Publisher
American Physiological Society
Online
2019-11-28
DOI
10.1152/ajpcell.00425.2019
References
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Note: Only part of the references are listed.- Complete steric exclusion of ions and proton transport through confined monolayer water
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- (2017) Wenlin Zhang et al. EBioMedicine
- Mouse Slc4a11 expressed in Xenopus oocytes is an ideally selective H+/OH− conductance pathway that is stimulated by rises in intracellular and extracellular pH
- (2016) Evan J. Myers et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Salt-Excluding Artificial Water Channels Exhibiting Enhanced Dipolar Water and Proton Translocation
- (2016) Erol Licsandru et al. JOURNAL OF THE AMERICAN CHEMICAL SOCIETY
- Human SLC4A11-C functions as a DIDS-stimulatable H+(OH−) permeation pathway: partial correction of R109H mutant transport
- (2015) Liyo Kao et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Missense mutation inSLC4A11in two Pakistani families affected with congenital hereditary endothelial dystrophy (CHED2)
- (2015) Haiba Kaul et al. Clinical and Experimental Optometry
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- (2015) Wenlin Zhang et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Crystal structure of the anion exchanger domain of human erythrocyte band 3
- (2015) T. Arakawa et al. SCIENCE
- Biosynthetic and functional defects in newly identified SLC4A11 mutants and absence of COL8A2 mutations in Fuchs endothelial corneal dystrophy
- (2014) Nagasamy Soumittra et al. JOURNAL OF HUMAN GENETICS
- Transmembrane water-flux through SLC4A11: a route defective in genetic corneal diseases
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- The Molecular Basis of K+Exclusion by theEscherichia coliAmmonium Channel AmtB
- (2013) Jason A. Hall et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Transient formation of water-conducting states in membrane transporters
- (2013) J. Li et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Missense mutations in the sodium borate cotransporter SLC4A11 cause late-onset Fuchs corneal dystrophya
- (2010) S. Amer Riazuddin et al. HUMAN MUTATION
- SLC4A11 Prevents Osmotic Imbalance Leading to Corneal Endothelial Dystrophy, Deafness, and Polyuria
- (2010) Nicole Gröger et al. JOURNAL OF BIOLOGICAL CHEMISTRY
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