Hypoxia and ER stress promote Staufen1 expression through an alternative translation mechanism

Title
Hypoxia and ER stress promote Staufen1 expression through an alternative translation mechanism
Authors
Keywords
Stau1 (Staufen 1), IRES (internal ribosome entry site), Hypoxia, Endoplasmic reticulum stress, UPR (unfolded protein response), PERK (PKR-Like endoplasmic reticulum kinase)
Journal
Publisher
Elsevier BV
Online
2016-09-18
DOI
10.1016/j.bbrc.2016.09.082

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