Phosphatase PPM1L Prevents Excessive Inflammatory Responses and Cardiac Dysfunction after Myocardial Infarction by Inhibiting IKKβ Activation
Published 2019 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Phosphatase PPM1L Prevents Excessive Inflammatory Responses and Cardiac Dysfunction after Myocardial Infarction by Inhibiting IKKβ Activation
Authors
Keywords
-
Journal
JOURNAL OF IMMUNOLOGY
Volume 203, Issue 5, Pages 1338-1347
Publisher
The American Association of Immunologists
Online
2019-08-20
DOI
10.4049/jimmunol.1900148
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- DUSP14 knockout accelerates cardiac ischemia reperfusion (IR) injury through activating NF-κB and MAPKs signaling pathways modulated by ROS generation
- (2018) Bin Lin et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Protein tyrosine phosphatases in cardiac physiology and pathophysiology
- (2018) Fallou Wade et al. HEART FAILURE REVIEWS
- Acute myocardial infarction
- (2017) Grant W Reed et al. LANCET
- IRF3 and type I interferons fuel a fatal response to myocardial infarction
- (2017) Kevin R King et al. NATURE MEDICINE
- Genomics and evolution of protein phosphatases
- (2017) Mark J. Chen et al. Science Signaling
- Genomics and evolution of protein phosphatases
- (2017) Mark J. Chen et al. Science Signaling
- DUSP8 Regulates Cardiac Ventricular Remodeling by Altering ERK1/2 SignalingNovelty and Significance
- (2016) Ruijie Liu et al. CIRCULATION RESEARCH
- The Biological Basis for Cardiac Repair After Myocardial Infarction
- (2016) Sumanth D. Prabhu et al. CIRCULATION RESEARCH
- The crossroads of inflammation, fibrosis, and arrhythmia following myocardial infarction
- (2016) Samantha D. Francis Stuart et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Inflammation as a Driver of Adverse Left Ventricular Remodeling After Acute Myocardial Infarction
- (2016) Peter C. Westman et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- The emerging role of Toll-like receptor 4 in myocardial inflammation
- (2016) Y Yang et al. Cell Death & Disease
- MCP-1-induced protein attenuates post-infarct cardiac remodeling and dysfunction through mitigating NF-κB activation and suppressing inflammation-associated microRNA expression
- (2015) Jianli Niu et al. BASIC RESEARCH IN CARDIOLOGY
- Inflammation in cardiac injury, repair and regeneration
- (2015) Nikolaos G. Frangogiannis CURRENT OPINION IN CARDIOLOGY
- Overexpression of protein phosphatase 2A in a murine model of chronic myocardial infarction leads to increased adverse remodeling but restores the regulation of β-catenin by glycogen synthase kinase 3β
- (2015) M. Hoehn et al. INTERNATIONAL JOURNAL OF CARDIOLOGY
- Myocardial infarction: a critical role of macrophages in cardiac remodeling
- (2015) Tobias Weinberger et al. Frontiers in Physiology
- Cardiac macrophages and their role in ischaemic heart disease
- (2014) Stefan Frantz et al. CARDIOVASCULAR RESEARCH
- Cardiac Fibroblast Glycogen Synthase Kinase-3β Regulates Ventricular Remodeling and Dysfunction in Ischemic Heart
- (2014) Hind Lal et al. CIRCULATION
- Inflammation and its Resolution as Determinants of Acute Coronary Syndromes
- (2014) Peter Libby et al. CIRCULATION RESEARCH
- Enhanced angiogenesis and increased cardiac perfusion after myocardial infarction in protein tyrosine phosphatase 1B-deficient mice
- (2014) Marie Besnier et al. FASEB JOURNAL
- Post-Infarction Inflammation and Left Ventricular Remodeling
- (2013) Toshihisa Anzai CIRCULATION JOURNAL
- Targeting inflammatory pathways in myocardial infarction
- (2013) Panagiota Christia et al. EUROPEAN JOURNAL OF CLINICAL INVESTIGATION
- Function and regulation of serine/threonine phosphatases in the healthy and diseased heart
- (2013) Jordi Heijman et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- PPM1l encodes an inositol requiring-protein 1 (IRE1) specific phosphatase that regulates the functional outcome of the ER stress response
- (2013) Gang Lu et al. Molecular Metabolism
- Phosphatase PTEN is critically involved in post-myocardial infarction remodeling through the Akt/interleukin-10 signaling pathway
- (2012) Nirmal Parajuli et al. BASIC RESEARCH IN CARDIOLOGY
- The innate immune response in reperfused myocardium
- (2012) L. Timmers et al. CARDIOVASCULAR RESEARCH
- Acyl-CoA binding domain containing 3 (ACBD3) recruits the protein phosphatase PPM1L to ER-Golgi membrane contact sites
- (2012) Yasuharu Shinoda et al. FEBS LETTERS
- IKK biology
- (2012) Fei Liu et al. IMMUNOLOGICAL REVIEWS
- Deficiency ofCapn4Gene Inhibits Nuclear Factor-κB (NF-κB) Protein Signaling/Inflammation and Reduces Remodeling after Myocardial Infarction
- (2012) Jian Ma et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Reduction of heart failure by pharmacological inhibition or gene deletion of protein tyrosine phosphatase 1B
- (2012) Elodie Gomez et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Anti-inflammatory mechanisms and therapeutic opportunities in myocardial infarct healing
- (2012) Tibor Kempf et al. JOURNAL OF MOLECULAR MEDICINE-JMM
- Intracellular MHC class II molecules promote TLR-triggered innate immune responses by maintaining activation of the kinase Btk
- (2011) Xingguang Liu et al. NATURE IMMUNOLOGY
- Innate immune signaling in cardiac ischemia
- (2011) Fatih Arslan et al. Nature Reviews Cardiology
- PHLPP-1 Negatively Regulates Akt Activity and Survival in the Heart
- (2010) Shigeki Miyamoto et al. CIRCULATION RESEARCH
- Serine/Threonine Phosphatases: Mechanism through Structure
- (2009) Yigong Shi CELL
- Macrophage roles following myocardial infarction
- (2008) Jessica M. Lambert et al. INTERNATIONAL JOURNAL OF CARDIOLOGY
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started