Sema3E is required for migration of cranial neural crest cells in zebrafish: Implications for the pathogenesis of CHARGE syndrome
Published 2019 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Sema3E is required for migration of cranial neural crest cells in zebrafish: Implications for the pathogenesis of CHARGE syndrome
Authors
Keywords
-
Journal
INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY
Volume -, Issue -, Pages -
Publisher
Wiley
Online
2019-08-29
DOI
10.1111/iep.12331
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Chd7 Is Critical for Early T-Cell Development and Thymus Organogenesis in Zebrafish
- (2018) Zhi-Zhi Liu et al. AMERICAN JOURNAL OF PATHOLOGY
- Sema3a plays a role in the pathogenesis of CHARGE syndrome
- (2018) Roser Ufartes et al. HUMAN MOLECULAR GENETICS
- Dysregulation of cotranscriptional alternative splicing underlies CHARGE syndrome
- (2018) Catherine Bélanger et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- CHARGE Syndrome
- (2017) Alexandra Hudson et al. PEDIATRICS IN REVIEW
- CHARGE syndrome modeling using patient-iPSCs reveals defective migration of neural crest cells harboring CHD7 mutations
- (2017) Hironobu Okuno et al. eLife
- Response to correspondence to Hale et al. atypical phenotypes associated with pathogenicCHD7variants and a proposal for broadening CHARGE syndrome clinical diagnostic criteria
- (2016) Caitlin L. Hale et al. AMERICAN JOURNAL OF MEDICAL GENETICS PART A
- A critical role for the chromatin remodeller CHD7 in anterior mesoderm during cardiovascular development
- (2015) Sophie Payne et al. DEVELOPMENTAL BIOLOGY
- Semaphorin signalling during development
- (2014) B. C. Jongbloets et al. DEVELOPMENT
- CHD7, the gene mutated in CHARGE syndrome, regulates genes involved in neural crest cell guidance
- (2014) Yvonne Schulz et al. HUMAN GENETICS
- Inappropriate p53 activation during development induces features of CHARGE syndrome
- (2014) Jeanine L. Van Nostrand et al. NATURE
- cAMP-Induced Expression of Neuropilin1 Promotes Retinal Axon Crossing in the Zebrafish Optic Chiasm
- (2013) A. L. Dell et al. JOURNAL OF NEUROSCIENCE
- Dynamic control of 1 integrin adhesion by the plexinD1-sema3E axis
- (2013) Y. I. Choi et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Brg1 governs distinct pathways to direct multiple aspects of mammalian neural crest cell development
- (2013) W. Li et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Molecular and phenotypic aspects ofCHD7mutation in CHARGE syndrome
- (2010) Gabriel E. Zentner et al. AMERICAN JOURNAL OF MEDICAL GENETICS PART A
- Chromodomain proteins in development: lessons from CHARGE syndrome
- (2010) WS Layman et al. CLINICAL GENETICS
- Sema3E–Plexin D1 signaling drives human cancer cell invasiveness and metastatic spreading in mice
- (2010) Andrea Casazza et al. JOURNAL OF CLINICAL INVESTIGATION
- The Calmodulin-Stimulated Adenylate Cyclase ADCY8 Sets the Sensitivity of Zebrafish Retinal Axons to Midline Repellents and Is Required for Normal Midline Crossing
- (2010) H. Xu et al. JOURNAL OF NEUROSCIENCE
- CHD7 cooperates with PBAF to control multipotent neural crest formation
- (2010) Ruchi Bajpai et al. NATURE
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started