Interaction between 17β-estradiol, prolactin and human papillomavirus induce E6/E7 transcript and modulate the expression and localization of hormonal receptors
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Title
Interaction between 17β-estradiol, prolactin and human papillomavirus induce E6/E7 transcript and modulate the expression and localization of hormonal receptors
Authors
Keywords
17β-estradiol, Prolactin, HPV, E6/E7 oncogenes, ERα, ERβ, GPER, PRLR
Journal
Cancer Cell International
Volume 19, Issue 1, Pages -
Publisher
Springer Science and Business Media LLC
Online
2019-09-03
DOI
10.1186/s12935-019-0935-6
References
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- Prolactin Activates ERα in the Absence of Ligand in Female Mammary Development and Carcinogenesis in Vivo
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- Prolactin and estradiol utilize distinct mechanisms to increase serine-118 phosphorylation and decrease levels of estrogen receptor α in T47D breast cancer cells
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- Activation of the unliganded estrogen receptor by prolactin in breast cancer cells
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