Maternal High-Fat Diet Causes a Sex-Dependent Increase in AGTR2 Expression and Cardiac Dysfunction in Adult Male Rat Offspring1
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Title
Maternal High-Fat Diet Causes a Sex-Dependent Increase in AGTR2 Expression and Cardiac Dysfunction in Adult Male Rat Offspring1
Authors
Keywords
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Journal
BIOLOGY OF REPRODUCTION
Volume 93, Issue 2, Pages -
Publisher
Oxford University Press (OUP)
Online
2015-07-09
DOI
10.1095/biolreprod.115.129916
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Note: Only part of the references are listed.- Sexual Dimorphism in Offspring Glucose-Sensitive Hypothalamic Gene Expression and Physiological Responses to Maternal High-Fat Diet Feeding
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- Prevalence of Obesity and Trends in the Distribution of Body Mass Index Among US Adults, 1999-2010
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- Conserved methylation of the glucocorticoid receptor gene exon 17 promoter in rats subjected to a maternal methyl-supplemented diet
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- Prenatal Hypoxia Causes a Sex-Dependent Increase in Heart Susceptibility to Ischemia and Reperfusion Injury in Adult Male Offspring: Role of Protein Kinase C
- (2009) Q. Xue et al. JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS
- Effect of In Utero and Early-Life Conditions on Adult Health and Disease
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- Highly individual methylation patterns of alternative glucocorticoid receptor promoters suggest individualized epigenetic regulatory mechanisms
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- Prenatal diet determines susceptibility to cardiac ischaemia–reperfusion injury following treatment with diethylmaleic acid and N-acetylcysteine
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