Infertility in Female Mice with a Gain-of-Function Mutation in the Luteinizing Hormone Receptor Is Due to Irregular Estrous Cyclicity, Anovulation, Hormonal Alterations, and Polycystic Ovaries1

Precocious Puberty and Leydig Cell Hyperplasia in Male Mice With a Gain of Function Mutation in the LH Receptor Gene

(2013) Stacey R. McGee et al.   ENDOCRINOLOGY

Ovulation Involves the Luteinizing Hormone-Dependent Activation of Gq/11in Granulosa Cells

(2013) Shawn M. Breen et al.   MOLECULAR ENDOCRINOLOGY

Reproductive and Metabolic Phenotype of a Mouse Model of PCOS

(2012) E. Leonie A.F. van Houten et al.   ENDOCRINOLOGY

Animal models of polycystic ovary syndrome: a focused review of rodent models in relationship to clinical phenotypes and cardiometabolic risk

(2012) Danni Shi et al.   FERTILITY AND STERILITY

Mutation Analysis of the LH Receptor Gene in Leydig Cell Adenoma and Hyperplasia and Functional and Biochemical Studies of Activating Mutations of the LH Receptor Gene

(2011) Annemieke M. Boot et al.   JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM

Novel signaling mechanisms in the ovary during oocyte maturation and ovulation

(2011) Marco Conti et al.   MOLECULAR AND CELLULAR ENDOCRINOLOGY

Female Mice Expressing Constitutively Active Mutants of FSH Receptor Present with a Phenotype of Premature Follicle Depletion and Estrogen Excess

(2010) Hellevi Peltoketo et al.   ENDOCRINOLOGY

The ovary: basic biology and clinical implications

(2010) JoAnne S. Richards et al.   JOURNAL OF CLINICAL INVESTIGATION

MAPK3/1 (ERK1/2) in Ovarian Granulosa Cells Are Essential for Female Fertility

(2009) H.-Y. Fan et al.   SCIENCE