Loss of Satb2 in the Cortex and Hippocampus Leads to Abnormal Behaviors in Mice
Published 2019 View Full Article
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Title
Loss of Satb2 in the Cortex and Hippocampus Leads to Abnormal Behaviors in Mice
Authors
Keywords
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Journal
Frontiers in Molecular Neuroscience
Volume 12, Issue -, Pages -
Publisher
Frontiers Media SA
Online
2019-02-12
DOI
10.3389/fnmol.2019.00033
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Note: Only part of the references are listed.- Natural history and genotype-phenotype correlations in 72 individuals with SATB2 -associated syndrome
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- SATB2-associated syndrome: Mechanisms, phenotype, and practical recommendations
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- Satb2 Is Required for the Development of a Spinal Exteroceptive Microcircuit that Modulates Limb Position
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- Mutual regulation betweenSatb2andFezf2promotes subcerebral projection neuron identity in the developing cerebral cortex
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- Expression of Transcription Factor Satb2 in Adult Mouse Brain
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- Overproduction of Upper-Layer Neurons in the Neocortex Leads to Autism-like Features in Mice
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- Interstitial Duplication of 2q32.1-q33.3 in a Patient With Epilepsy, Developmental Delay, and Autistic Behavior
- (2013) Daisuke Usui et al. AMERICAN JOURNAL OF MEDICAL GENETICS PART A
- Impaired cliff avoidance reaction in dopamine transporter knockout mice
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- The Prevalence of DSM-IV Attention-Deficit/Hyperactivity Disorder: A Meta-Analytic Review
- (2012) Erik G. Willcutt Neurotherapeutics
- Adult Raphe-Specific Deletion of Lmx1b Leads to Central Serotonin Deficiency
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- Satb2 Is a Postmitotic Determinant for Upper-Layer Neuron Specification in the Neocortex
- (2008) Olga Britanova et al. NEURON
- Satb2 Regulates Callosal Projection Neuron Identity in the Developing Cerebral Cortex
- (2008) Elizabeth A. Alcamo et al. NEURON
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