KATP channel deficiency in mouse FDB causes an impairment of energy metabolism during fatigue

Contractile abnormalities of mouse muscles expressing hyperkalemic periodic paralysis mutant NaV1.4 channels do not correlate with Na+ influx or channel content

(2014) Brooke Lucas et al.   PHYSIOLOGICAL GENOMICS

Kir6.2 limits Ca2+ overload and mitochondrial oscillations of ventricular myocytes in response to metabolic stress

(2013) Nina M. Storey et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

Sarcolemmal ATP-sensitive potassium channels modulate skeletal muscle function under low-intensity workloads

(2013) Zhiyong Zhu et al.   JOURNAL OF GENERAL PHYSIOLOGY

The KATP channel Kir6.2 subunit content is higher in glycolytic than oxidative skeletal muscle fibers

(2011) Krystyna Banas et al.   AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY

Sarcolemmal ATP-Sensitive K+ Channels Control Energy Expenditure Determining Body Weight

(2010) Alexey E. Alekseev et al.   Cell Metabolism

Naturally occurring R225W mutation of the gene encoding AMP-activated protein kinase (AMPK)γ3 results in increased oxidative capacity and glucose uptake in human primary myotubes

(2010) S. A. Crawford et al.   DIABETOLOGIA

Fatigue preconditioning increases fatigue resistance in mouse flexor digitorum brevis muscles with non-functioning KATPchannels

(2010) Louise Boudreault et al.   JOURNAL OF PHYSIOLOGY-LONDON

Impaired exercise tolerance and skeletal muscle myopathy in sulfonylurea receptor-2 mutant mice

(2009) Douglas Stoller et al.   AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY

Comparison of regulated passive membrane conductance in action potential–firing fast- and slow-twitch muscle

(2009) Thomas Holm Pedersen et al.   JOURNAL OF GENERAL PHYSIOLOGY

Regulation of ClC-1 and KATPchannels in action potential–firing fast-twitch muscle fibers

(2009) Thomas Holm Pedersen et al.   JOURNAL OF GENERAL PHYSIOLOGY

Contractile dysfunctions in ATP-dependent K+channel-deficient mouse muscle during fatigue involve excessive depolarization and Ca2+influx through L-type Ca2+channels

(2008) Carlo Cifelli et al.   EXPERIMENTAL PHYSIOLOGY

Muscle K+, Na+, and Cl− disturbances and Na+-K+ pump inactivation: implications for fatigue

(2007) Michael J. McKenna et al.   JOURNAL OF APPLIED PHYSIOLOGY