MicroRNA-495-3p inhibits multidrug resistance by modulating autophagy through GRP78/mTOR axis in gastric cancer
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Title
MicroRNA-495-3p inhibits multidrug resistance by modulating autophagy through GRP78/mTOR axis in gastric cancer
Authors
Keywords
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Journal
Cell Death & Disease
Volume 9, Issue 11, Pages -
Publisher
Springer Nature America, Inc
Online
2018-10-19
DOI
10.1038/s41419-018-0950-x
References
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Related references
Note: Only part of the references are listed.- miR-495 sensitizes MDR cancer cells to the combination of doxorubicin and taxol by inhibiting MDR1 expression
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- Autophagy inhibition re-sensitizes pulse stimulation-selected paclitaxel-resistant triple negative breast cancer cells to chemotherapy-induced apoptosis
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- Induction of autophagy counteracts the anticancer effect of cisplatin in human esophageal cancer cells with acquired drug resistance
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- MicroRNAs, miR-154, miR-299-5p, miR-376a, miR-376c, miR-377, miR-381, miR-487b, miR-485-3p, miR-495 and miR-654-3p, mapped to the 14q32.31 locus, regulate proliferation, apoptosis, migration and invasion in metastatic prostate cancer cells
- (2013) A Formosa et al. ONCOGENE
- miR-495 and miR-551a inhibit the migration and invasion of human gastric cancer cells by directly interacting with PRL-3
- (2012) Zhengrong Li et al. CANCER LETTERS
- Reduced miR-128 in Breast Tumor-Initiating Cells Induces Chemotherapeutic Resistance via Bmi-1 and ABCC5
- (2011) Y. Zhu et al. CLINICAL CANCER RESEARCH
- MicroRNA-30a Sensitizes Tumor Cells to cis-Platinum via Suppressing Beclin 1-mediated Autophagy
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- AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1
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- Autophagy and the Integrated Stress Response
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- Autophagy in the Pathogenesis of Disease
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