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Quorum sensing controls thePseudomonas aeruginosaCRISPR-Cas adaptive immune system

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Mechanisms of bacterial persistence during stress and antibiotic exposure

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Interference-driven spacer acquisition is dominant over naive and primed adaptation in a native CRISPR–Cas system

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Parasite Exposure Drives Selective Evolution of Constitutive versus Inducible Defense

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Pseudomonas aeruginosa Biofilm Infections: Community Structure, Antimicrobial Tolerance and Immune Response

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Regulation of the Type I-F CRISPR-Cas system by CRP-cAMP and GalM controls spacer acquisition and interference

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Multidimensional metrics for estimating phage abundance, distribution, gene density, and sequence coverage in metagenomes

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Adaptation in bacterial CRISPR-Cas immunity can be driven by defective phages

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Type I-E CRISPR-Cas Systems Discriminate Target from Non-Target DNA through Base Pairing-Independent PAM Recognition

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The CRISPR/Cas Adaptive Immune System of Pseudomonas aeruginosa Mediates Resistance to Naturally Occurring and Engineered Phages

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Molecular memory of prior infections activates the CRISPR/Cas adaptive bacterial immunity system

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RNA-guided complex from a bacterial immune system enhances target recognition through seed sequence interactions

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Pseudomonas aeruginosa Increases Formation of Multidrug-Tolerant Persister Cells in Response to Quorum-Sensing Signaling Molecules

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The CRISPR/Cas bacterial immune system cleaves bacteriophage and plasmid DNA

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Transcription Profile of Thermus thermophilus CRISPR Systems after Phage Infection

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Small CRISPR RNAs Guide Antiviral Defense in Prokaryotes

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CRISPR Interference Limits Horizontal Gene Transfer in Staphylococci by Targeting DNA

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Nongenetic Individuality in the Host–Phage Interaction

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