MiR-20b targets AKT3 and modulates vascular endothelial growth factor-mediated changes in diabetic retinopathy

Odontoblastic Differentiation, Inflammatory Response, and Angiogenic Potential of 4 Calcium Silicate–based Cements: Micromega MTA, ProRoot MTA, RetroMTA, and Experimental Calcium Silicate Cement

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Contribution of epigenetics in diabetic retinopathy

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Akt isoforms in vascular disease

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AKT3 promotes prostate cancer proliferation cells through regulation of Akt, B-Raf & TSC1/TSC2

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MicroRNA-424 inhibits Akt3/E2F3 axis and tumor growth in hepatocellular carcinoma

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Flotillin-2 promotes metastasis of nasopharyngeal carcinoma by activating NF-κB and PI3K/Akt3 signaling pathways

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Vitamin D manipulates miR-181c, miR-20b and miR-15a in human umbilical vein endothelial cells exposed to a diabetic-like environment

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MicroRNA-22 Induces Endothelial Progenitor Cell Senescence by Targeting AKT3

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Role of Endothelial Dysfunction and Arterial Stiffness in the Development of Diabetic Retinopathy: Figure 1

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Role of microRNAs in the modulation of diabetic retinopathy

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The Role of MicroRNAs in Diabetic Nephropathy

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MicroRNA in Diabetic Nephropathy: Renin Angiotensin, AGE/RAGE, and Oxidative Stress Pathway

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Papel do fator de crescimento vascular endotelial na angiogênese e na retinopatia diabética

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MicroRNA-200b Regulates Vascular Endothelial Growth Factor-Mediated Alterations in Diabetic Retinopathy

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miR-146a–Mediated Extracellular Matrix Protein Production in Chronic Diabetes Complications

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Akt3 controls vascular endothelial growth factor secretion and angiogenesis in ovarian cancer cells

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Modulation of microRNA 20b with resveratrol and longevinex is linked with their potent anti-angiogenic action in the ischaemic myocardium and synergestic effects of resveratrol and γ-tocotrienol

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Oxidative Stress and Diabetic Complications

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Plasma MicroRNA Profiling Reveals Loss of Endothelial MiR-126 and Other MicroRNAs in Type 2 Diabetes

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Transcriptional coactivator p300 regulates glucose-induced gene expression in endothelial cells

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Microvascular lesions of diabetic retinopathy: clues towards understanding pathogenesis?

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