Journal
NEUROSCIENCE LETTERS
Volume 698, Issue -, Pages 217-223Publisher
ELSEVIER IRELAND LTD
DOI: 10.1016/j.neulet.2019.01.034
Keywords
Somatostatin; Cell excitability; SSTR2; K-ir channel; Desensitization
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Funding
- Friedrich-Baur-Stiftung [03/16]
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Many neurological diseases are related to disturbances of somatostatin- (SOM-) expressing interneurons in the cingulate cortex. Therefore, their role within the circuitry of the cingulate cortex needs to be investigated. We describe here the physiological time course of SOM effects onto pyramidal cell excitability and action potential discharge pattern. Furthermore, we show that the GRK2 inhibitor Gallein had no effect on the reduced SOM-induced response following repetitive SOM applications.
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