Group 2 innate lymphoid cells (ILC2) are regulated by stem cell factor during chronic asthmatic disease
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Title
Group 2 innate lymphoid cells (ILC2) are regulated by stem cell factor during chronic asthmatic disease
Authors
Keywords
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Journal
Mucosal Immunology
Volume -, Issue -, Pages -
Publisher
Springer Nature
Online
2019-01-08
DOI
10.1038/s41385-018-0117-1
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- (2014) Mihalis Verykokakis et al. IMMUNOLOGICAL REVIEWS
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- (2013) Taylor A. Doherty et al. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
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- (2012) P. M. O'Byrne et al. CLINICAL AND EXPERIMENTAL ALLERGY
- The Transcription Factor GATA-3 Controls Cell Fate and Maintenance of Type 2 Innate Lymphoid Cells
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- (2012) Bryan C Petersen et al. NATURE MEDICINE
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- (2011) Yuejuan Li et al. JOURNAL OF EXPERIMENTAL MEDICINE
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- (2010) Steven A. Saenz et al. NATURE
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- (2009) V. Dolgachev et al. JOURNAL OF IMMUNOLOGY
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- (2007) Vladislav Dolgachev et al. AMERICAN JOURNAL OF PATHOLOGY
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