Obesity-associated family with sequence similarity 13, member A (FAM13A) is dispensable for adipose development and insulin sensitivity
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Title
Obesity-associated family with sequence similarity 13, member A (FAM13A) is dispensable for adipose development and insulin sensitivity
Authors
Keywords
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Journal
INTERNATIONAL JOURNAL OF OBESITY
Volume -, Issue -, Pages -
Publisher
Springer Nature America, Inc
Online
2018-10-09
DOI
10.1038/s41366-018-0222-y
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Note: Only part of the references are listed.- Family with sequence similarity 13, member A modulates adipocyte insulin signaling and preserves systemic metabolic homeostasis
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- A Chronic Obstructive Pulmonary Disease Susceptibility Gene,FAM13A, Regulates Protein Stability of β-Catenin
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- Numerous Genes in Loci Associated With Body Fat Distribution Are Linked to Adipose Function
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- FAM13A is associated with non-small cell lung cancer (NSCLC) progression and controls tumor cell proliferation and survival
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- New genetic loci link adipose and insulin biology to body fat distribution
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- Genetic studies of body mass index yield new insights for obesity biology
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- Moving beyond genetics: isFAM13Aa major biological contributor in lung physiology and chronic lung diseases?
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- The Human Lipodystrophy Gene Product Berardinelli-Seip Congenital Lipodystrophy 2/Seipin Plays a Key Role in Adipocyte Differentiation
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