Expression of pro-inflammatory cytokines and mediators induced by Bisphenol A via ERK-NFκB and JAK1/2-STAT3 pathways in macrophages
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Title
Expression of pro-inflammatory cytokines and mediators induced by Bisphenol A via ERK-NFκB and JAK1/2-STAT3 pathways in macrophages
Authors
Keywords
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Journal
ENVIRONMENTAL TOXICOLOGY
Volume -, Issue -, Pages -
Publisher
Wiley
Online
2019-01-05
DOI
10.1002/tox.22702
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Note: Only part of the references are listed.- Ecotoxicological evaluation of the risk posed by bisphenol A, triclosan, and 4-nonylphenol in coastal waters using early life stages of marine organisms ( Isochrysis galbana, Mytilus galloprovincialis, Paracentrotus lividus, and Acartia clausi )
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- Wogonin attenuates endotoxin-induced prostaglandin E2 and nitric oxide production via Src-ERK1/2-NFκB pathway in BV-2 microglial cells
- (2013) Chung-Hsin Yeh et al. ENVIRONMENTAL TOXICOLOGY
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- Bisphenol-A Impairs Insulin Action and Up-Regulates Inflammatory Pathways in Human Subcutaneous Adipocytes and 3T3-L1 Cells
- (2013) Rossella Valentino et al. PLoS One
- Protective effect of wogonin on proinflammatory cytokine generation via Jak1/3-STAT1/3 pathway in lipopolysaccharide stimulated BV2 microglial cells
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- Proinflammatory activation of macrophages by bisphenol A-glycidyl-methacrylate involved NFκB activation via PI3K/Akt pathway
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- Bisphenol A (BPA) in China: A review of sources, environmental levels, and potential human health impacts
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- Ubiquitination and Degradation of the Inhibitors of NF- B
- (2009) N. Kanarek et al. Cold Spring Harbor Perspectives in Biology
- Suppression of NO production and 8-nitroguanosine formation by phenol-containing endocrine-disrupting chemicals in LPS-stimulated macrophages: Involvement of estrogen receptor-dependent or -independent pathways
- (2008) Jun Yoshitake et al. NITRIC OXIDE-BIOLOGY AND CHEMISTRY
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