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Title
Gap junctions–guards of excitability
Authors
Keywords
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Journal
BIOCHEMICAL SOCIETY TRANSACTIONS
Volume 43, Issue 3, Pages 508-512
Publisher
Portland Press Ltd.
Online
2015-05-26
DOI
10.1042/bst20150059
References
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- Missense Mutations in Plakophilin-2 Cause Sodium Current Deficit and Associate With a Brugada Syndrome Phenotype
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- Managing the complexity of communication: regulation of gap junctions by post-translational modification
- (2013) Lene N. Axelsen et al. Frontiers in Pharmacology
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- Remodeling of the cardiac sodium channel, connexin43, and plakoglobin at the intercalated disk in patients with arrhythmogenic cardiomyopathy
- (2012) Maartje Noorman et al. HEART RHYTHM
- Cx43 Associates with Nav1.5 in the Cardiomyocyte Perinexus
- (2012) J. Matthew Rhett et al. JOURNAL OF MEMBRANE BIOLOGY
- Reduced heterogeneous expression of Cx43 results in decreased Nav1.5 expression and reduced sodium current that accounts for arrhythmia vulnerability in conditional Cx43 knockout mice
- (2011) John A. Jansen et al. HEART RHYTHM
- Connexin 43 connexon to gap junction transition is regulated by zonula occludens-1
- (2011) J. Matthew Rhett et al. MOLECULAR BIOLOGY OF THE CELL
- Connexin43 Modulates Cell Polarity and Directional Cell Migration by Regulating Microtubule Dynamics
- (2011) Richard Francis et al. PLoS One
- SAP97 and Dystrophin Macromolecular Complexes Determine Two Pools of Cardiac Sodium Channels Na v 1.5 in Cardiomyocytes
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- (2008) John S. Lowe et al. JOURNAL OF CELL BIOLOGY
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