CO2carbamylation of proteins as a mechanism in physiology

Connexin26 hemichannels with a mutation that causes KID syndrome in humans lack sensitivity to CO2

(2014) Louise Meigh et al.   eLife

CO2 directly modulates connexin 26 by formation of carbamate bridges between subunits

(2013) Louise Meigh et al.   eLife

The role of connexins in ear and skin physiology — Functional insights from disease-associated mutations

(2012) Ji Xu et al.   BIOCHIMICA ET BIOPHYSICA ACTA-BIOMEMBRANES

A novel mutation in the connexin 26 gene (GJB2) in a child with clinical and histological features of keratitis-ichthyosis-deafness (KID) syndrome

(2010) U. Koppelhus et al.   CLINICAL AND EXPERIMENTAL DERMATOLOGY

Severe form of keratitis-ichthyosis-deafness (KID) syndrome associated with septic complications

(2010) Kunitaka HARUNA et al.   JOURNAL OF DERMATOLOGY

CO2-dependent opening of connexin 26 and related β connexins

(2010) Robert T. R. Huckstepp et al.   JOURNAL OF PHYSIOLOGY-LONDON

Connexin hemichannel-mediated CO2-dependent release of ATP in the medulla oblongata contributes to central respiratory chemosensitivity

(2010) Robert T. R. Huckstepp et al.   JOURNAL OF PHYSIOLOGY-LONDON

Structure of the connexin 26 gap junction channel at 3.5 Å resolution

(2009) Shoji Maeda et al.   NATURE