Malformations in the Murine Kidney Caused by Loss of CENP-F Function
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Title
Malformations in the Murine Kidney Caused by Loss of CENP-F Function
Authors
Keywords
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Journal
Anatomical Record-Advances in Integrative Anatomy and Evolutionary Biology
Volume 302, Issue 1, Pages 163-170
Publisher
Wiley
Online
2018-11-09
DOI
10.1002/ar.24018
References
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Related references
Note: Only part of the references are listed.- The ischemic/nephrotoxic acute kidney injury and the use of renal biomarkers in clinical practice
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- Loss of CENP-F results in distinct microtubule-related defects without chromosomal abnormalities
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- Centromere protein F includes two sites that couple efficiently to depolymerizing microtubules
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- The snRNA-processing complex, Integrator, is required for ciliogenesis and dynein recruitment to the nuclear envelope via distinct mechanisms
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- Urinary Biomarkers in the Clinical Prognosis and Early Detection of Acute Kidney Injury
- (2010) J. L. Koyner et al. Clinical Journal of the American Society of Nephrology
- Genetic kidney diseases
- (2010) Friedhelm Hildebrandt LANCET
- PPARγ and chronic kidney disease
- (2010) Agnes B. Fogo PEDIATRIC NEPHROLOGY
- Urinary Biomarkers in the Early Detection of Acute Kidney Injury after Cardiac Surgery
- (2009) W. K. Han et al. Clinical Journal of the American Society of Nephrology
- Uremic memory: the role of acute kidney injury in long-term outcomes
- (2009) Ladan Golestaneh et al. KIDNEY INTERNATIONAL
- Murine CENP-F Regulates Centrosomal Microtubule Nucleation and Interacts with Hook2 at the Centrosome
- (2009) Katherine L. Moynihan et al. MOLECULAR BIOLOGY OF THE CELL
- Murine CENPF interacts with syntaxin 4 in the regulation of vesicular transport
- (2008) R. D. Pooley et al. JOURNAL OF CELL SCIENCE
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