MUTYH, an adenine DNA glycosylase, mediates p53 tumor suppression via PARP-dependent cell death

Host Immune Defense Peptide LL-37 Activates Caspase-Independent Apoptosis and Suppresses Colon Cancer

(2012) S. X. Ren et al.   CANCER RESEARCH

8-Oxoguanine causes neurodegeneration during MUTYH-mediated DNA base excision repair

(2012) Zijing Sheng et al.   JOURNAL OF CLINICAL INVESTIGATION

Involvement of Werner syndrome protein in MUTYH-mediated repair of oxidative DNA damage

(2012) Radhakrishnan Kanagaraj et al.   NUCLEIC ACIDS RESEARCH

Oxidative Stress Induces Nuclear-to-Cytosol Shift of hMSH3, a Potential Mechanism for EMAST in Colorectal Cancer Cells

(2012) Stephanie S. Tseng-Rogenski et al.   PLoS One

DNA glycosylase encoded by MUTYH functions as a molecular switch for programmed cell death under oxidative stress to suppress tumorigenesis

(2011) Sugako Oka et al.   CANCER SCIENCE

p73 protein regulates DNA damage repair

(2011) Elena Zaika et al.   FASEB JOURNAL

Leiden open variation database of the MUTYH gene

(2010) Astrid A. Out et al.   HUMAN MUTATION

Genetic Instability Caused by Loss of MutS Homologue 3 in Human Colorectal Cancer

(2008) A. C. Haugen et al.   CANCER RESEARCH

No death without life: vital functions of apoptotic effectors

(2008) L Galluzzi et al.   CELL DEATH AND DIFFERENTIATION

Two distinct pathways of cell death triggered by oxidative damage to nuclear and mitochondrial DNAs

(2008) Sugako Oka et al.   EMBO JOURNAL

Transcriptional control of human p53-regulated genes

(2008) Todd Riley et al.   NATURE REVIEWS MOLECULAR CELL BIOLOGY