YAP drives cutaneous squamous cell carcinoma formation and progression
Published 2018 View Full Article
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Title
YAP drives cutaneous squamous cell carcinoma formation and progression
Authors
Keywords
-
Journal
eLife
Volume 7, Issue -, Pages -
Publisher
eLife Sciences Publications, Ltd
Online
2018-09-20
DOI
10.7554/elife.33304
References
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Note: Only part of the references are listed.- YAP and TAZ are essential for basal and squamous cell carcinoma initiation
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- Distinct modes of SMAD2 chromatin binding and remodeling shape the transcriptional response to NODAL/Activin signaling
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- Integrin signalling regulates YAP and TAZ to control skin homeostasis
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- ZEB1 turns into a transcriptional activator by interacting with YAP1 in aggressive cancer types
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- Adhesion to fibronectin regulates Hippo signaling via the FAK–Src–PI3K pathway
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- Genome-wide association between YAP/TAZ/TEAD and AP-1 at enhancers drives oncogenic growth
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- Fra-1/AP-1 induces EMT in mammary epithelial cells by modulating Zeb1/2 and TGFβ expression
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- Mutational landscape of gingivo-buccal oral squamous cell carcinoma reveals new recurrently-mutated genes and molecular subgroups
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- Yap1 Acts Downstream of α-Catenin to Control Epidermal Proliferation
- (2011) Karin Schlegelmilch et al. CELL
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- (2011) Sirio Dupont et al. NATURE
- Yes-associated protein (YAP) transcriptional coactivator functions in balancing growth and differentiation in skin
- (2011) H. Zhang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- TGF-beta1 causes epithelial-mesenchymal transition in HaCaT derivatives, but induces expression of COX-2 and migration only in benign, not in malignant keratinocytes
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- Epithelial-Mesenchymal Transition: At the Crossroads of Development and Tumor Metastasis
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