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Title
Animal Toxins Can Alter the Function of Nav1.8 and Nav1.9
Authors
Keywords
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Journal
Toxins
Volume 4, Issue 8, Pages 620-632
Publisher
MDPI AG
Online
2012-08-14
DOI
10.3390/toxins4080620
References
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Related references
Note: Only part of the references are listed.- Functional Analysis of Stably Expressed Human Nav1.9
- (2012) Carlos G. Vanoye et al. BIOPHYSICAL JOURNAL
- Biophysical properties of Nav1.8/Nav1.2 chimeras and inhibition by µO-conotoxin MrVIB
- (2012) O Knapp et al. BRITISH JOURNAL OF PHARMACOLOGY
- Functional Na V 1.8 Channels in Intracardiac Neurons
- (2012) Arie O. Verkerk et al. CIRCULATION RESEARCH
- Identification of New Batrachotoxin-sensing Residues in Segment IIIS6 of the Sodium Channel
- (2011) Yuzhe Du et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Functional properties and toxin pharmacology of a dorsal root ganglion sodium channel viewed through its voltage sensors
- (2011) Frank Bosmans et al. JOURNAL OF GENERAL PHYSIOLOGY
- Menthol pain relief through cumulative inactivation of voltage-gated sodium channels
- (2011) Christelle Gaudioso et al. PAIN
- Nav1.9 Channel Contributes to Mechanical and Heat Pain Hypersensitivity Induced by Subacute and Chronic Inflammation
- (2011) Stéphane Lolignier et al. PLoS One
- Sodium Channels in Normal and Pathological Pain
- (2010) Sulayman D. Dib-Hajj et al. Annual Review of Neuroscience
- Cooccupancy of the Outer Vestibule of Voltage-Gated Sodium Channels by μ-Conotoxin KIIIA and Saxitoxin or Tetrodotoxin
- (2010) Min-Min Zhang et al. JOURNAL OF NEUROPHYSIOLOGY
- The Tetrodotoxin Binding Site Is within the Outer Vestibule of the Sodium Channel
- (2010) Harry A. Fozzard et al. Marine Drugs
- Targeting voltage sensors in sodium channels with spider toxins
- (2010) Frank Bosmans et al. TRENDS IN PHARMACOLOGICAL SCIENCES
- Exploring the role of nociceptor-specific sodium channels in pain transmission using Nav1.8 and Nav1.9 knockout mice
- (2009) Sandra Leo et al. BEHAVIOURAL BRAIN RESEARCH
- Activation of neurokinin 3 receptor increases Nav1.9 current in enteric neurons
- (2009) Carine Copel et al. JOURNAL OF PHYSIOLOGY-LONDON
- Interactions between lipids and voltage sensor paddles detected with tarantula toxins
- (2009) Mirela Milescu et al. NATURE STRUCTURAL & MOLECULAR BIOLOGY
- Jingzhaotoxin-IX, a novel gating modifier of both sodium and potassium channels from Chinese tarantula Chilobrachys jingzhao
- (2009) Meichun Deng et al. NEUROPHARMACOLOGY
- The voltage-gated sodium channel Nav1.9 is required for inflammation-based urinary bladder dysfunction
- (2009) Amy M. Ritter et al. NEUROSCIENCE LETTERS
- Lack of colonic inflammation-induced acute visceral hypersensitivity to colorectal distension in Nav1.9 knockout mice
- (2008) V. Martinez et al. EUROPEAN JOURNAL OF PAIN
- Tetrodotoxin-resistant voltage-gated sodium channels Nav1.8 and Nav1.9 are expressed in the retina
- (2008) Brendan J. O'Brien et al. JOURNAL OF COMPARATIVE NEUROLOGY
- Inflammatory Mediators Increase Nav1.9 Current and Excitability in Nociceptors through a Coincident Detection Mechanism
- (2008) François Maingret et al. JOURNAL OF GENERAL PHYSIOLOGY
- Deconstructing voltage sensor function and pharmacology in sodium channels
- (2008) Frank Bosmans et al. NATURE
- Structural Dynamics of an Isolated Voltage-Sensor Domain in a Lipid Bilayer
- (2008) Sudha Chakrapani et al. STRUCTURE
- GTP up-regulated persistent Na+current and enhanced nociceptor excitability require NaV1.9
- (2007) Johan A. R. Östman et al. JOURNAL OF PHYSIOLOGY-LONDON
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