The NLRP3 inflammasome activation in human or mouse cells, sensitivity causes puzzle
Published 2013 View Full Article
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Title
The NLRP3 inflammasome activation in human or mouse cells, sensitivity causes puzzle
Authors
Keywords
Mouse Cell, NLRP3 Inflammasome, Pyrin, NLRP3 INFLAMMASOME Activation, Primary Monocyte
Journal
Protein & Cell
Volume 4, Issue 8, Pages 565-568
Publisher
Springer Nature
Online
2013-06-20
DOI
10.1007/s13238-013-3905-0
References
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Related references
Note: Only part of the references are listed.- Acute lipopolysaccharide priming boosts inflammasome activation independently of inflammasome sensor induction
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- (2012) Y. He et al. JOURNAL OF IMMUNOLOGY
- Deubiquitination of NLRP3 by BRCC3 Critically Regulates Inflammasome Activity
- (2012) Bénédicte F. Py et al. MOLECULAR CELL
- Conservation and divergence in Toll-like receptor 4-regulated gene expression in primary human versus mouse macrophages
- (2012) K. Schroder et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- GBP5 Promotes NLRP3 Inflammasome Assembly and Immunity in Mammals
- (2012) A. R. Shenoy et al. SCIENCE
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- The NLRC4 inflammasome receptors for bacterial flagellin and type III secretion apparatus
- (2011) Yue Zhao et al. NATURE
- Innate immune recognition of bacterial ligands by NAIPs determines inflammasome specificity
- (2011) Eric M. Kofoed et al. NATURE
- The Inflammasomes
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- New insights into the nature of autoinflammatory diseases from mice withNlrp3mutations
- (2010) Guangxun Meng et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- Aspergillus fumigatus Stimulates the NLRP3 Inflammasome through a Pathway Requiring ROS Production and the Syk Tyrosine Kinase
- (2010) Najwane Saïd-Sadier et al. PLoS One
- A Mutation in the Nlrp3 Gene Causing Inflammasome Hyperactivation Potentiates Th17 Cell-Dominant Immune Responses
- (2009) Guangxun Meng et al. IMMUNITY
- Differential requirement for the activation of the inflammasome for processing and release of IL-1 in monocytes and macrophages
- (2008) M. G. Netea et al. BLOOD
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