Crimean-Congo Hemorrhagic Fever Virus Entry into Host Cells Occurs through the Multivesicular Body and Requires ESCRT Regulators

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Crimean–Congo hemorrhagic fever virus utilizes a clathrin- and early endosome-dependent entry pathway

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Identification of a putative Crimean-Congo hemorrhagic fever virus entry factor

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Old World Arenaviruses Enter the Host Cell via the Multivesicular Body and Depend on the Endosomal Sorting Complex Required for Transport

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Microtubule-dependent and microtubule-independent steps in Crimean-Congo hemorrhagic fever virus replication cycle

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Crimean-Congo hemorrhagic fever virus entry and replication is clathrin-, pH- and cholesterol-dependent

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The ESCRT-I Subunit TSG101 Controls Endosome-to-Cytosol Release of Viral RNA

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Oropouche virus entry into HeLa cells involves clathrin and requires endosomal acidification

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