Knock-In Reporter Mice Demonstrate that DNA Repair by Non-homologous End Joining Declines with Age
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Title
Knock-In Reporter Mice Demonstrate that DNA Repair by Non-homologous End Joining Declines with Age
Authors
Keywords
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Journal
PLoS Genetics
Volume 10, Issue 7, Pages e1004511
Publisher
Public Library of Science (PLoS)
Online
2014-07-18
DOI
10.1371/journal.pgen.1004511
References
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Related references
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- (2013) L. N. Truong et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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- (2011) Julie Della-Maria et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- SIRT6 Promotes DNA Repair Under Stress by Activating PARP1
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- (2010) Michael R. Lieber Annual Review of Biochemistry
- Functions of astrocytes and their potential as therapeutic targets
- (2010) Harold K. Kimelberg et al. Neurotherapeutics
- Synthesis-dependent microhomology-mediated end joining accounts for multiple types of repair junctions
- (2010) Amy Marie Yu et al. NUCLEIC ACIDS RESEARCH
- Looking Older
- (2008) Gary J. Fisher et al. ARCHIVES OF DERMATOLOGY
- DNA double-strand breaks: A potential causative factor for mammalian aging?
- (2008) Han Li et al. MECHANISMS OF AGEING AND DEVELOPMENT
- DNA damage and ageing: new-age ideas for an age-old problem
- (2008) George A. Garinis et al. NATURE CELL BIOLOGY
- Effect of Ku80 Deficiency on Mutation Frequencies and Spectra at a LacZ Reporter Locus in Mouse Tissues and Cells
- (2008) Rita A. Busuttil et al. PLoS One
- MMEJ repair of double-strand breaks (director’s cut): deleted sequences and alternative endings
- (2008) Mitch McVey et al. TRENDS IN GENETICS
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