Genetic Models of Apoptosis-Induced Proliferation Decipher Activation of JNK and Identify a Requirement of EGFR Signaling for Tissue Regenerative Responses in Drosophila
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Title
Genetic Models of Apoptosis-Induced Proliferation Decipher Activation of JNK and Identify a Requirement of EGFR Signaling for Tissue Regenerative Responses in Drosophila
Authors
Keywords
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Journal
PLoS Genetics
Volume 10, Issue 1, Pages e1004131
Publisher
Public Library of Science (PLoS)
Online
2014-01-31
DOI
10.1371/journal.pgen.1004131
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Note: Only part of the references are listed.- In Drosophila, RhoGEF2 cooperates with activated Ras in tumorigenesis through a pathway involving Rho1-Rok-Myosin-II and JNK signalling
- (2013) P. Khoo et al. Disease Models & Mechanisms
- Caspase signalling in the absence of apoptosis drives Jnk-dependent invasion
- (2013) Vivek A Rudrapatna et al. EMBO REPORTS
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- (2012) B Mollereau et al. CELL DEATH AND DIFFERENTIATION
- A Versatile ΦC31 Based Reporter System for Measuring AP-1 and Nrf2 Signaling in Drosophila and in Tissue Culture
- (2012) Nirmalya Chatterjee et al. PLoS One
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- (2012) Kenneth D. Irvine SEMINARS IN CELL & DEVELOPMENTAL BIOLOGY
- The Role of Apoptosis-Induced Proliferation for Regeneration and Cancer
- (2012) H. D. Ryoo et al. Cold Spring Harbor Perspectives in Biology
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- (2011) E Shlevkov et al. CELL DEATH AND DIFFERENTIATION
- Injury-induced activation of the MAPK/CREB pathway triggers apoptosis-induced compensatory proliferation in hydra head regeneration
- (2011) Simona Chera et al. DEVELOPMENT GROWTH & DIFFERENTIATION
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- (2011) Brent S. Wells et al. DEVELOPMENTAL BIOLOGY
- Identification of Novel Ras-Cooperating Oncogenes inDrosophila melanogaster: A RhoGEF/Rho-Family/JNK Pathway Is a Central Driver of Tumorigenesis
- (2011) Anthony M. Brumby et al. GENETICS
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- (2011) Abigail R. Gerhold et al. GENETICS
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- (2011) Yaron Suissa et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Caspase 3–mediated stimulation of tumor cell repopulation during cancer radiotherapy
- (2011) Qian Huang et al. NATURE MEDICINE
- The Cdc42/Par6/aPKC Polarity Complex Regulates Apoptosis-Induced Compensatory Proliferation in Epithelia
- (2010) Stephen J. Warner et al. CURRENT BIOLOGY
- Cell death-induced regeneration in wing imaginal discs requires JNK signalling
- (2010) C. Bergantinos et al. DEVELOPMENT
- Hippo signaling: growth control and beyond
- (2010) G. Halder et al. DEVELOPMENT
- Regulation of Hippo signaling by Jun kinase signaling during compensatory cell proliferation and regeneration, and in neoplastic tumors
- (2010) Gongping Sun et al. DEVELOPMENTAL BIOLOGY
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- (2010) Clayton M. Morrison et al. GENESIS
- A Targeted UAS-RNAi Screen in Drosophila Larvae Identifies Wound Closure Genes Regulating Distinct Cellular Processes
- (2010) C. Lesch et al. GENETICS
- Rho1 regulates apoptosis via activation of the JNK signaling pathway at the plasma membrane
- (2010) Amanda L. Neisch et al. JOURNAL OF CELL BIOLOGY
- CD95 promotes tumour growth
- (2010) Lina Chen et al. NATURE
- Apoptosis, Stem Cells, and Tissue Regeneration
- (2010) A. Bergmann et al. Science Signaling
- Correcting developmental errors by apoptosis: lessons from Drosophila JNK signaling
- (2009) Tatsushi Igaki APOPTOSIS
- The cleaved-Caspase-3 antibody is a marker of Caspase-9-like DRONC activity in Drosophila
- (2009) Y Fan et al. CELL DEATH AND DIFFERENTIATION
- A Blood-Borne PDGF/VEGF-like Ligand Initiates Wound-Induced Epidermal Cell Migration in Drosophila Larvae
- (2009) Yujane Wu et al. CURRENT BIOLOGY
- Cytokine Signaling Mediates UV-Induced Nociceptive Sensitization in Drosophila Larvae
- (2009) Daniel T. Babcock et al. CURRENT BIOLOGY
- The role of Dpp and Wg in compensatory proliferation and in the formation of hyperplastic overgrowths caused by apoptotic cells in the Drosophila wing disc
- (2009) A. Perez-Garijo et al. DEVELOPMENT
- Cell death and tissue remodeling in planarian regeneration
- (2009) Jason Pellettieri et al. DEVELOPMENTAL BIOLOGY
- Regenerative Growth in Drosophila Imaginal Discs Is Regulated by Wingless and Myc
- (2009) Rachel K. Smith-Bolton et al. DEVELOPMENTAL CELL
- Apoptotic Cells Provide an Unexpected Source of Wnt3 Signaling to Drive Hydra Head Regeneration
- (2009) Simona Chera et al. DEVELOPMENTAL CELL
- Apoptosis and cancer: mutations within caspase genes
- (2009) S Ghavami et al. JOURNAL OF MEDICAL GENETICS
- A Non-Redundant Role for Drosophila Mkk4 and Hemipterous/Mkk7 in TAK1-Mediated Activation of JNK
- (2009) Peter Geuking et al. PLoS One
- Activation mechanism and substrate specificity of the Drosophila initiator caspase DRONC
- (2008) S J Snipas et al. CELL DEATH AND DIFFERENTIATION
- Distinct Mechanisms of Apoptosis-Induced Compensatory Proliferation in Proliferating and Differentiating Tissues in the Drosophila Eye
- (2008) Yun Fan et al. DEVELOPMENTAL CELL
- Apoptosis-induced compensatory proliferation. The Cell is dead. Long live the Cell!
- (2008) Yun Fan et al. TRENDS IN CELL BIOLOGY
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