Reduced Na+ and higher K+ channel expression and function contribute to right ventricular origin of arrhythmias in Scn5a+/- mice
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Title
Reduced Na+ and higher K+ channel expression and function contribute to right ventricular origin of arrhythmias in Scn5a+/- mice
Authors
Keywords
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Journal
Open Biology
Volume 2, Issue 6, Pages 120072-120072
Publisher
The Royal Society
Online
2012-06-14
DOI
10.1098/rsob.120072
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Related references
Note: Only part of the references are listed.- Mapping of reentrant spontaneous polymorphic ventricular tachycardia in a Scn5a+/− mouse model
- (2011) Claire A. Martin et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Re-Evaluation of the Action Potential Upstroke Velocity as a Measure of the Na+ Current in Cardiac Myocytes at Physiological Conditions
- (2011) Géza Berecki et al. PLoS One
- Increased Right Ventricular Repolarization Gradients Promote Arrhythmogenesis in a Murine Model of Brugada Syndrome
- (2010) CLAIRE A. MARTIN et al. JOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY
- In vivo studies of Scn5a+/− mice modeling Brugada syndrome demonstrate both conduction and repolarization abnormalities
- (2010) Claire A. Martin et al. JOURNAL OF ELECTROCARDIOLOGY
- Variable Nav1.5 Protein Expression from the Wild-Type Allele Correlates with the Penetrance of Cardiac Conduction Disease in the Scn5a+/− Mouse Model
- (2010) Anne-Laure Leoni et al. PLoS One
- The cardiac sodium channel displays differential distribution in the conduction system and transmural heterogeneity in the murine ventricular myocardium
- (2009) C. A. Remme et al. BASIC RESEARCH IN CARDIOLOGY
- Calcium-dependent regulation of voltage-gated sodium channels in cardiac myocytes: just the beginning?
- (2009) K. H. Yuill et al. CARDIOVASCULAR RESEARCH
- Combined reduction of intercellular coupling and membrane excitability differentially affects transverse and longitudinal cardiac conduction
- (2009) M. Stein et al. CARDIOVASCULAR RESEARCH
- Scn3b knockout mice exhibit abnormal ventricular electrophysiological properties
- (2009) Parvez Hakim et al. PROGRESS IN BIOPHYSICS & MOLECULAR BIOLOGY
- The complexity of genotype-phenotype relations associated with loss-of-function sodium channel mutations and the role of in silico studies
- (2008) Arthur A. M. Wilde et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Cytoskeletal Protein 4.1R Affects Repolarization and Regulates Calcium Handling in the Heart
- (2008) Mark A. Stagg et al. CIRCULATION RESEARCH
- Transcriptional profiling of ion channel genes in Brugada syndrome and other right ventricular arrhythmogenic diseases
- (2008) Nathalie Gaborit et al. EUROPEAN HEART JOURNAL
- Post-transcriptional gene silencing of KChIP2 and Navβ1 in neonatal rat cardiac myocytes reveals a functional association between Na and Ito currents
- (2008) Isabelle Deschênes et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Effect of heterogeneous APD restitution on VF organization in a model of the human ventricles
- (2007) R. H. Keldermann et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- The cardiac persistent sodium current: an appealing therapeutic target?
- (2007) D A Saint BRITISH JOURNAL OF PHARMACOLOGY
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