Axonal degeneration in an Alzheimer mouse model is PS1 gene dose dependent and linked to intraneuronal Aβ accumulation
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Title
Axonal degeneration in an Alzheimer mouse model is PS1 gene dose dependent and linked to intraneuronal Aβ accumulation
Authors
Keywords
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Journal
Frontiers in Aging Neuroscience
Volume 6, Issue -, Pages -
Publisher
Frontiers Media SA
Online
2014-06-27
DOI
10.3389/fnagi.2014.00139
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Note: Only part of the references are listed.- Amyloid- Peptide Oligomers Disrupt Axonal Transport through an NMDA Receptor-Dependent Mechanism That Is Mediated by Glycogen Synthase Kinase 3 in Primary Cultured Hippocampal Neurons
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- Transient intraneuronal Aβ rather than extracellular plaque pathology correlates with neuron loss in the frontal cortex of APP/PS1KI mice
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