Constitutive activation of CREB in mice enhances temporal association learning and increases hippocampal CA1 neuronal spine density and complexity

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Hippocampal Metaplasticity Is Required for the Formation of Temporal Associative Memories

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Neurons Are Recruited to a Memory Trace Based on Relative Neuronal Excitability Immediately before Training

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CREB regulates spine density of lateral amygdala neurons: implications for memory allocation

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Enhanced intrinsic excitability and EPSP-spike coupling accompany enriched environment-induced facilitation of LTP in hippocampal CA1 pyramidal neurons

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Reduced Spine Density in Specific Regions of CA1 Pyramidal Neurons in Two Transgenic Mouse Models of Alzheimer's Disease

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Upregulation of CREB-Mediated Transcription Enhances Both Short- and Long-Term Memory

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Physiological variability in brain-derived neurotrophic factor expression predicts dendritic spine density in the mouse dentate gyrus

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Lipocalin-2 controls neuronal excitability and anxiety by regulating dendritic spine formation and maturation

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Entorhinal Cortex Layer III Input to the Hippocampus Is Crucial for Temporal Association Memory

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CaMKIV over-expression boosts cortical 4-7 Hz oscillations during learning and 1-4 Hz delta oscillations during sleep

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The Postsynaptic Density Proteins Homer and Shank Form a Polymeric Network Structure

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An activity-induced microRNA controls dendritic spine formation by regulating Rac1-PAK signaling

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CREB regulates excitability and the allocation of memory to subsets of neurons in the amygdala

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Hippocampal CA3 Output Is Crucial for Ripple-Associated Reactivation and Consolidation of Memory

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Endocytic Trafficking and Recycling Maintain a Pool of Mobile Surface AMPA Receptors Required for Synaptic Potentiation

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Selective Erasure of a Fear Memory

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Upregulation of Calcium/Calmodulin-Dependent Protein Kinase IV Improves Memory Formation and Rescues Memory Loss with Aging

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Automated Three-Dimensional Detection and Shape Classification of Dendritic Spines from Fluorescence Microscopy Images

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Transgenic Inhibition of Synaptic Transmission Reveals Role of CA3 Output in Hippocampal Learning

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