NOGO-A/RTN4A and NOGO-B/RTN4B are simultaneously expressed in epithelial, fibroblast and neuronal cells and maintain ER morphology

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Endoplasmic Protein Nogo-B (RTN4-B) Interacts with GRAMD4 and Regulates TLR9-Mediated Innate Immune Responses

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Rab18 and a Rab18 GEF complex are required for normal ER structure

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Hereditary spastic paraplegia proteins REEP1, spastin, and atlastin-1 coordinate microtubule interactions with the tubular ER network

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Reticulon Short Hairpin Transmembrane Domains Are Used to Shape ER Tubules

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Reticulon 4B (Nogo-B) is necessary for macrophage infiltration and tissue repair

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Nucleic Acid Binding of the RTN1-C C-Terminal Region: Toward the Functional Role of a Reticulon Protein†

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The Reticulon and Dp1/Yop1p Proteins Form Immobile Oligomers in the Tubular Endoplasmic Reticulum

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Membrane Proteins of the Endoplasmic Reticulum Induce High-Curvature Tubules

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