Therapeutically targeting mitochondrial redox signalling alleviates endothelial dysfunction in preeclampsia
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Title
Therapeutically targeting mitochondrial redox signalling alleviates endothelial dysfunction in preeclampsia
Authors
Keywords
-
Journal
Scientific Reports
Volume 6, Issue 1, Pages -
Publisher
Springer Nature
Online
2016-09-08
DOI
10.1038/srep32683
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Related references
Note: Only part of the references are listed.- Immunostimulatory role of mitochondrial DAMPs: alarming for pre-eclampsia?
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- Excessive autophagy induces the failure of trophoblast invasion and vasculature
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- Nitroso-Redox Balance and Mitochondrial Homeostasis Are Regulated by STOX1, a Pre-Eclampsia-Associated Gene
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- Diagnosis, evaluation, and management of the hypertensive disorders of pregnancy
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- Peroxisome Proliferator-activated Receptor γ, Coactivator 1α Deletion Induces Angiotensin II–Associated Vascular Dysfunction by Increasing Mitochondrial Oxidative Stress and Vascular Inflammation
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- Mitochondrial Biogenesis through Activation of Nuclear Signaling Proteins
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- Robust Early Pregnancy Prediction of Later Preeclampsia Using Metabolomic Biomarkers
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- Circulating mitochondrial DAMPs cause inflammatory responses to injury
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- Decidual expression and maternal serum levels of heme oxygenase 1 are increased in pre-eclampsia
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